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The Journal of Clinical Investigation|December 5, 2017
STAT5BN642H is a driver mutation for T cell neoplasiaHa Thi Thanh Pham, Barbara Maurer, Michaela Prchal-Murphy, et al.
Leukemia|February 24, 2018
Pharmacologic inhibition of STAT5 in acute myeloid leukemiaBettina Wingelhofer, Barbara Maurer, Elizabeth C Heyes, et al.
Cell Death & Disease|October 14, 2016
Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformationTahereh Javaheri, Zahra Kazemi, Jan Pencik, et al.
Cell Death & Disease|July 25, 2018
Correction: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformationTahereh Javaheri, Zahra Kazemi, Jan Pencik, et al.
Cell Death & Disease|August 14, 2019
Retraction Note: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformationTahereh Javaheri, Zahra Kazemi, Jan Pencik, et al.
American Journal of Human Genetics|April 23, 2019
A Specific CNOT1 Mutation Results in a Novel Syndrome of Pancreatic Agenesis and Holoprosencephaly through Impaired Pancreatic and Neurological DevelopmentElisa De Franco, Rachel A Watson, Wolfgang J Weninger, et al.
Blood|April 29, 2020
CDK6 is an essential direct target of NUP98 fusion proteins in acute myeloid leukemiaJohannes Schmoellerl, Inês Amorim Monteiro Barbosa, Thomas Eder, et al.
Nature Immunology|April 24, 2024
JAK-STAT signaling maintains homeostasis in T cells and macrophagesNikolaus Fortelny, Matthias Farlik, Victoria Fife, et al.
Haematologica|May 25, 2019
High activation of STAT5A drives peripheral T-cell lymphoma and leukemiaBarbara Maurer, Harini Nivarthi, Bettina Wingelhofer, et al.
The Journal of Clinical Investigation|April 15, 2024
Hyperactive STAT5 hijacks T cell receptor signaling and drives immature T cell acute lymphoblastic leukemiaTobias Suske, Helena Sorger, Gabriele Manhart, et al.
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