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The Journal of Clinical Investigation|November 3, 2010
T-cadherin is critical for adiponectin-mediated cardioprotection in miceMartin S Denzel, Maria-Cecilia Scimia, Philine M Zumstein, et al.
Brain Structure & Function|April 8, 2017
Protective role of Cadherin 13 in interneuron developmentAbigail C Killen, Melissa Barber, Joshua J W Paulin, et al.
Cancer Research|March 5, 2008
T-cadherin supports angiogenesis and adiponectin association with the vasculature in a mouse mammary tumor modelLionel W Hebbard, Michèle Garlatti, Lawrence J T Young, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 22, 2009
Lewis(x) and alpha2,3-sialyl glycans and their receptors TAG-1, Contactin, and L1 mediate CD24-dependent neurite outgrowthAnnika Lieberoth, Frauke Splittstoesser, Nainesh Katagihallimath, et al.
The European Journal of Neuroscience|July 21, 2005
Contactin regulates the current density and axonal expression of tetrodotoxin-resistant but not tetrodotoxin-sensitive sodium channels in DRG neuronsAnthony M Rush, Matthew J Craner, Takashi Kageyama, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 19, 2011
TrkB (tropomyosin-related kinase B) controls the assembly and maintenance of GABAergic synapses in the cerebellar cortexAlbert I Chen, Cindy N Nguyen, David R Copenhagen, et al.
Physiology & Behavior|June 15, 2007
Evidence for hypothalamic dysregulation in mouse models of anorexia as well as in humansJeanette E Johansen, Sergueï O Fetissov, Ulrika Bergström, et al.
Plos One|June 12, 2013
Role of the adiponectin binding protein, T-cadherin (cdh13), in pulmonary responses to subacute ozoneDavid I Kasahara, Alison S Williams, Leandro A Benedito, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 6, 2010
Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowthWilliam J Brackenbury, Jeffrey D Calhoun, Chunling Chen, et al.
Molecular and Cellular Biology|July 25, 2007
Deletion of Shp2 in the brain leads to defective proliferation and differentiation in neural stem cells and early postnatal lethalityYuehai Ke, Eric E Zhang, Kazuki Hagihara, et al.
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