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Practical Laboratory Medicine|January 8, 2020
Evaluation of a semi-automatic isoelectric focusing method for apolipoprotein E phenotypingRanda Bittar, Alain Carrié, Georges Nouadje, et al.
Journal of Inherited Metabolic Disease|February 25, 2011
Short-chain 3-hydroxyacyl-CoA dehydrogenase deficiency: the clinical relevance of an early diagnosis and report of four new casesEsmeralda Martins, M Luis Cardoso, Esmeralda Rodrigues, et al.
American Journal of Public Health|April 15, 2016
Estimating Potential Reductions in Premature Mortality in New York City From Raising the Minimum Wage to $15Tsu-Yu Tsao, Kevin J Konty, Gretchen Van Wye, et al.
British Journal of Haematology|November 12, 2005
Erythropoietin levels in the different clinical forms of hereditary spherocytosisS Rocha, E Costa, C Catarino, et al.
Acta Medica Portuguesa|March 9, 2000
[Glucose-6-phosphate dehydrogenase deficiency in 2 girls]E Costa, J M Cabeda, M E Abreu, et al.
British Journal of Haematology|October 29, 2000
A new PKLR gene mutation in the R-type promoter region affects the gene transcription causing pyruvate kinase deficiencyL Manco, M L Ribeiro, V Máximo, et al.
Journal of Clinical Pathology|May 1, 1993
Use of plasma iodine assay for diagnosing thyroid disordersP Allain, S Berre, N Krari, et al.
The Journal of Clinical Endocrinology and Metabolism|January 1, 1994
Pentagastrin stimulation test and early diagnosis of medullary thyroid carcinoma using an immunoradiometric assay of calcitonin: comparison with genetic screening in hereditary medullary thyroid carcinomaN Barbot, C Calmettes, I Schuffenecker, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 15, 2007
Uncoupling protein-2 controls proliferation by promoting fatty acid oxidation and limiting glycolysis-derived pyruvate utilizationClaire Pecqueur, Thi Bui, Chantal Gelly, et al.
Annales D'Endocrinologie|January 1, 1997
Results of the calcitonin stimulation test in normal volunteers compared with genetically unaffected members of MEN 2A and familial medullary thyroid carcinoma familiesN Wion-Barbot, I Schuffenecker, P Niccoli, et al.
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