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Medical Hypotheses|August 3, 2010
Upregulation of N-acetylaspartic acid resulting nitric oxide toxicity induces aspartoacylase mutations and protein interaction to cause pathophysiology seen in Canavan diseaseSankar SurendranPhysical Chemistry Chemical Physics : PCCP|April 30, 2016
A symbiotic view of the origin of life at hydrothermal impact crater-lakesSankar ChatterjeeBMC Genomics|February 22, 2016
The effects of sample size on population genomic analyses--implications for the tests of neutralitySankar SubramanianMedical Anthropology Quarterly|December 23, 2004
Communication and miscommunication in informed consent to researchPamela SankarGenetics|January 12, 2013
Significance of population size on the fixation of nonsynonymous mutations in genes under varying levels of selection pressureSankar SubramanianAustralian Journal of General Practice|September 4, 2019
Management of post-surgical wounds in general practiceSankar SinhaMolecular Biology Reports|October 19, 2007
Upregulation of N-acetylaspartic acid alters inflammation, transcription and contractile associated protein levels in the stomach and smooth muscle contractilitySankar SurendranEuropean Journal of Human Genetics : EJHG|April 19, 2012
Quantifying harmful mutations in human populationsSankar SubramanianHuman Genetics|October 15, 2015
Europeans have a higher proportion of high‑frequency deleterious variants than AfricansSankar SubramanianPageof 8,421