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Brain Injury|July 10, 2003
Traumatic brain injury and its effects on synaptic plasticityBenedict C Albensi, Damir JanigroAmerican Journal of Alzheimer'S Disease and Other Dementias|November 24, 2004
Why do many NMDA antagonists fail, while others are safe and effective at blocking excitotoxicity associated with dementia and acute injury?Benedict C Albensi, Chiazor Igoechi, Damir Janigro, et al.Brain Research|January 17, 2004
Activation of long-term synaptic plasticity causes suppression of epileptiform activity in rat hippocampal slicesBenedict C Albensi, Gabrielle Ata, Erin Schmidt, et al.Current Pharmaceutical Design|November 30, 2007
The NMDA receptor/ion channel complex: a drug target for modulating synaptic plasticity and excitotoxicityBenedict C AlbensiDrug News & Perspectives|September 16, 2003
A comparison of drug treatment versus electrical stimulation for suppressing seizure activityBenedict C AlbensiDrug News & Perspectives|April 5, 2003
Tumor Necrosis Factor and Drug Delivery in the Blood-Brain BarrierBenedict C. AlbensiExpert Opinion on Drug Discovery|March 20, 2013
Can in vitro assessment provide relevant end points for cognitive drug programs?Benedict C AlbensiFrontiers in Cell and Developmental Biology|August 27, 2019
What Is Nuclear Factor Kappa B (NF-κB) Doing in and to the Mitochondrion?Benedict C AlbensiDrug News & Perspectives|April 5, 2003
Biological Mechanisms, Cerebrovascular Alterations and Pharmacological Targets in the Pathology of DementiaBenedict C. AlbensiInternational Review of Neurobiology|June 19, 2019
Dysfunction of mitochondria: Implications for Alzheimer's diseaseBenedict C AlbensiPageof 20