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Molecular Neurobiology|November 6, 2015
Mechanisms of Mitochondrial Dysfunction in Alzheimer's DiseaseChris Cadonic, Mohammad Golam Sabbir, Benedict C AlbensiCurrent Alzheimer Research|February 23, 2016
Traumatic Brain Injury as a Risk Factor for Alzheimer's Disease: Is Inflammatory Signaling a Key Player?Jelena Djordjevic, Mohammad Golam Sabbir, Benedict C AlbensiCells|August 27, 2021
Role of Nrf2 in Synaptic Plasticity and Memory in Alzheimer's DiseaseDon A Davies, Aida Adlimoghaddam, Benedict C AlbensiFrontiers in Molecular Neuroscience|August 24, 2016
Ammonia as a Potential Neurotoxic Factor in Alzheimer's DiseaseAida Adlimoghaddam, Mohammad G Sabbir, Benedict C AlbensiFrontiers in Cell and Developmental Biology|June 5, 2023
Loss of cholinergic receptor muscarinic 1 impairs cortical mitochondrial structure and function: implications in Alzheimer's diseaseMohammad Golam Sabbir, Mamiko Swanson, Benedict C AlbensiMolecular Neurobiology|June 2, 2021
The Effect of COVID-19 on NF-κB and Neurological Manifestations of DiseaseDon A Davies, Aida Adlimoghaddam, Benedict C AlbensiCNS Neuroscience & Therapeutics|July 31, 2018
A review of clinical treatment considerations of donepezil in severe Alzheimer's diseaseAida Adlimoghaddam, Melanie Neuendorff, Banibrata Roy, et al.Current Alzheimer Research|March 11, 2016
Protein Structural Analysis of Calbindin D28k Function and Dysregulation: Potential Competition Between Ca(2+) and Zn(2.)Sara Ibrahim Omar, Benedict C Albensi, Kathleen M GoughAmerican Journal of Alzheimer'S Disease and Other Dementias|November 24, 2004
Why do many NMDA antagonists fail, while others are safe and effective at blocking excitotoxicity associated with dementia and acute injury?Benedict C Albensi, Chiazor Igoechi, Damir Janigro, et al.Drug Discovery Today|June 22, 2025
Advancements and challenges in using AI for biomarker detection in early Alzheimer's diseaseIman Beheshti, Benedict C Albensi, Alex Freitas, et al.Pageof 8