Showing results (61-70 of 359) with videos related to
Sort By:
Pageof 36
The Journal of Biological Chemistry|February 27, 2008
BI-1 regulates endoplasmic reticulum Ca2+ homeostasis downstream of Bcl-2 family proteinsChunyan Xu, Wenjie Xu, Amy E Palmer, et al.Proceedings of the National Academy of Sciences of the United States of America|October 13, 2005
Method for targeting protein destruction by using a ubiquitin-independent, proteasome-mediated degradation pathwayShu-ichi Matsuzawa, Michael Cuddy, Toru Fukushima, et al.The Biochemical Journal|April 27, 2004
Heterotypic interactions among NACHT domains: implications for regulation of innate immune responsesJason S Damiano, Vasco Oliveira, Kate Welsh, et al.Biochemical and Biophysical Research Communications|March 17, 2007
Characterization of a TIR-like protein from Paracoccus denitrificansLieh Yoon Low, Takashi Mukasa, John C Reed, et al.The Journal of Biological Chemistry|March 23, 2013
Novel phosphorylation and ubiquitination sites regulate reactive oxygen species-dependent degradation of anti-apoptotic c-FLIP proteinRachel P Wilkie-Grantham, Shu-Ichi Matsuzawa, John C ReedCancer Research|September 11, 2014
PCTAIRE1 phosphorylates p27 and regulates mitosis in cancer cellsTeruki Yanagi, Maryla Krajewska, Shu-ichi Matsuzawa, et al.Journal of Biotechnology|November 9, 2011
Expression, purification, and characterization of recombinant NOD1 (NLRC1): A NLR family memberNadav Askari, Ricardo G Correa, Dayong Zhai, et al.Cancer Research|March 26, 2002
Bag1 proteins regulate growth and survival of ZR-75-1 human breast cancer cellsMasafumi Kudoh, Deborah A Knee, Shinichi Takayama, et al.Proceedings of the National Academy of Sciences of the United States of America|December 9, 2004
Bcl-2-mediated alterations in endoplasmic reticulum Ca2+ analyzed with an improved genetically encoded fluorescent sensorAmy E Palmer, Can Jin, John C Reed, et al.Oncogene|May 29, 2002
Dysregulation of apoptosis genes in hematopoietic malignanciesShinichi Kitada, Irene M Pedersen, Aaron D Schimmer, et al.Pageof 36