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EMBO Reports|June 20, 2012
Loss of fused in sarcoma (FUS) promotes pathological Tau splicingDenise Orozco, Sabina Tahirovic, Kristin Rentzsch, et al.The Journal of Biological Chemistry|April 19, 2012
Membrane orientation and subcellular localization of transmembrane protein 106B (TMEM106B), a major risk factor for frontotemporal lobar degenerationChristina M Lang, Katrin Fellerer, Benjamin M Schwenk, et al.Current Opinion in Neurobiology|November 12, 2015
An amyloid-like cascade hypothesis for C9orf72 ALS/FTDDieter Edbauer, Christian HaassJournal of Molecular Medicine (Berlin, Germany)|August 27, 2013
FUS-mediated alternative splicing in the nervous system: consequences for ALS and FTLDDenise Orozco, Dieter EdbauerScience (New York, N.Y.)|October 6, 2022
A perturbed network in neurodegenerationJean-Marc Gallo, Dieter EdbauerCell Communication and Signaling : CCS|April 30, 2014
Good guy or bad guy: the opposing roles of microRNA 125b in cancerJulia Banzhaf-Strathmann, Dieter EdbauerEMBO Reports|June 15, 2023
C9orf72 protein quality control by UBR5-mediated heterotypic ubiquitin chainsJulia Jülg, Dieter Edbauer, Christian BehrendsThe EMBO Journal|December 21, 2013
The FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomesBenjamin M Schwenk, Christina M Lang, Sebastian Hogl, et al.The Journal of Biological Chemistry|June 24, 2004
Co-expression of nicastrin and presenilin rescues a loss of function mutant of APH-1Dieter Edbauer, Christoph Kaether, Harald Steiner, et al.Pageof 11