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Biorxiv : the Preprint Server for Biology|July 15, 2025
HNF4α controls growth, identity and response to KRAS inhibition of invasive mucinous adenocarcinoma of the lungHeadtlove Essel Dadzie, Yangsook Song Green, Soledad Camolotto, et al.
The EMBO Journal|October 1, 2004
The Rb tumor suppressor is required for stress erythropoiesisBenjamin T Spike, Alexandra Dirlam, Benjamin C Dibling, et al.
Anti-Cancer Drugs|March 27, 2002
Nucleoside transport inhibitors, dipyridamole and p-nitrobenzylthioinosine, selectively potentiate the antitumor activity of NB1011Christopher R Boyer, Patricia L Karjian, Geoffrey M Wahl, et al.
Life Science Alliance|January 22, 2020
Nup93 regulates breast tumor growth by modulating cell proliferation and actin cytoskeleton remodelingSimone Bersini, Nikki K Lytle, Roberta Schulte, et al.
Seminars in Cancer Biology|August 26, 2014
The multifaceted role of the embryonic gene Cripto-1 in cancer, stem cells and epithelial-mesenchymal transitionMalgorzata Klauzinska, Nadia P Castro, Maria Cristina Rangel, et al.
NPJ Breast Cancer|September 14, 2024
Mcam stabilizes a luminal progenitor-like breast cancer cell state via Ck2 control and Src/Akt/Stat3 attenuationOzlen Balcioglu, Brooke L Gates, David W Freeman, et al.
Biorxiv : the Preprint Server for Biology|April 2, 2024
Mcam stabilizes luminal progenitor breast cancer phenotypes via Ck2 control and Src/Akt/Stat3 attenuationOzlen Balcioglu, Brooke L Gates, David W Freeman, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 21, 2007
Quantitative analyses reveal the importance of regulated Hdmx degradation for p53 activationYunyuan V Wang, Mark Wade, Eetsin Wong, et al.
Molecular Cell|June 7, 2002
c-Myc can induce DNA damage, increase reactive oxygen species, and mitigate p53 function: a mechanism for oncogene-induced genetic instabilityOmid Vafa, Mark Wade, Suzanne Kern, et al.
Molecular and Cellular Biology|December 13, 2006
Mouse mutants reveal that putative protein interaction sites in the p53 proline-rich domain are dispensable for tumor suppressionFranck Toledo, Crystal J Lee, Kurt A Krummel, et al.
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