Showing results (1-10 of 21) with videos related to

Sort By:
Pageof 3
American Journal of Physiology. Gastrointestinal and Liver Physiology|December 30, 2006
Regulation of Triglyceride Metabolism. III. Emerging role of lipid droplet protein ADFP in health and diseaseBenny Hung-Junn Chang, Lawrence Chan
EMBO Molecular Medicine|December 1, 2012
Sustained expression of the transcription factor GLIS3 is required for normal beta cell function in adultsYisheng Yang, Benny Hung-Junn Chang, Lawrence Chan
Journal of Cellular Biochemistry|July 23, 2003
Ubiquitin-dependent and -independent proteasomal degradation of apoB associated with endoplasmic reticulum and Golgi apparatus, respectively, in HepG2 cellsWei Liao, Benny Hung-Junn Chang, Michael Mancini, et al.
Journal of Lipid Research|April 29, 2010
Absence of adipose differentiation related protein upregulates hepatic VLDL secretion, relieves hepatosteatosis, and improves whole body insulin resistance in leptin-deficient miceBenny Hung-Junn Chang, Lan Li, Pradip Saha, et al.
Circulation Research|May 17, 2008
Deficiency of adipose differentiation-related protein impairs foam cell formation and protects against atherosclerosisAntoni Paul, Benny Hung-Junn Chang, Lan Li, et al.
Nucleic Acids Research|March 7, 2009
The Krüppel-like zinc finger protein Glis3 directly and indirectly activates insulin gene transcriptionYisheng Yang, Benny Hung-Junn Chang, Susan L Samson, et al.
Scientific Reports|January 20, 2017
PLIN2 is a Key Regulator of the Unfolded Protein Response and Endoplasmic Reticulum Stress Resolution in Pancreatic β CellsElaine Chen, Tsung Huang Tsai, Lan Li, et al.
Molecular and Cellular Biology|January 24, 2006
Protection against fatty liver but normal adipogenesis in mice lacking adipose differentiation-related proteinBenny Hung-Junn Chang, Lan Li, Antoni Paul, et al.
Endocrinology|July 4, 2009
The human lipodystrophy gene product Berardinelli-Seip congenital lipodystrophy 2/seipin plays a key role in adipocyte differentiationWeiqin Chen, Vijay K Yechoor, Benny Hung-Junn Chang, et al.
Atherosclerosis|September 5, 2002
Increased intracellular calcium transients by calmodulin antagonists differentially modulate tumor necrosis factor-alpha-induced E-selectin and ICAM-1 expressionKuang-Hua Chen, Benny Hung-Junn Chang, Patrick Younan, et al.
Pageof 3