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Benoit I Giasson

Showing results (71-80 of 174) with videos related to

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The Journal of Biological Chemistry|October 27, 2019
Impaired tau-microtubule interactions are prevalent among pathogenic tau variants arising from missense mutationsYuxing Xia, Zachary A Sorrentino, Justin D Kim, et al.
Acta Neuropathologica Communications|May 4, 2021
Robust α-synuclein pathology in select brainstem neuronal populations is a potential instigator of multiple system atrophyEthan W Hass, Zachary A Sorrentino, Grace M Lloyd, et al.
Molecular Neurodegeneration|May 20, 2018
Differential induction of mutant SOD1 misfolding and aggregation by tau and α-synuclein pathologyMichael C Pace, Guilian Xu, Susan Fromholt, et al.
Neuromolecular Medicine|May 30, 2025
Tau from SPAM Transgenic Mice Exhibit Potent Strain-Specific Prion-Like Seeding Properties Characteristic of Human Neurodegenerative DiseasesEthan D Smith, Giavanna Paterno, Brach M Bell, et al.
The Journal of Biological Chemistry|December 21, 2017
Distinct differences in prion-like seeding and aggregation between Tau protein variants provide mechanistic insights into tauopathiesKevin H Strang, Cara L Croft, Zachary A Sorrentino, et al.
Molecular Cell|June 3, 2014
A cellular system that degrades misfolded proteins and protects against neurodegenerationLili Guo, Benoit I Giasson, Alex Glavis-Bloom, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|November 14, 2008
Clinical and pathological characteristics of patients with leucine-rich repeat kinase-2 mutationsJason P Covy, Wuxing Yuan, Elisa A Waxman, et al.
The Journal of Cell Biology|May 5, 2004
Degradative organelles containing mislocalized alpha-and beta-synuclein proliferate in presenilin-1 null neuronsChristina A Wilson, Diane D Murphy, Benoit I Giasson, et al.
Plos One|April 3, 2018
Novel monoclonal antibodies targeting the microtubule-binding domain of human tauCara L Croft, Brenda D Moore, Yong Ran, et al.
Molecular and Cellular Neurosciences|June 4, 2018
Partial loss of ATP13A2 causes selective gliosis independent of robust lipofuscinosisSruti Rayaprolu, Yasin B Seven, John Howard, et al.
Pageof 18

Showing results (71-80 of 174) with videos related to

Sort By:
Pageof 18
The Journal of Biological Chemistry|October 27, 2019
Impaired tau-microtubule interactions are prevalent among pathogenic tau variants arising from missense mutationsYuxing Xia, Zachary A Sorrentino, Justin D Kim, et al.
Acta Neuropathologica Communications|May 4, 2021
Robust α-synuclein pathology in select brainstem neuronal populations is a potential instigator of multiple system atrophyEthan W Hass, Zachary A Sorrentino, Grace M Lloyd, et al.
Molecular Neurodegeneration|May 20, 2018
Differential induction of mutant SOD1 misfolding and aggregation by tau and α-synuclein pathologyMichael C Pace, Guilian Xu, Susan Fromholt, et al.
Neuromolecular Medicine|May 30, 2025
Tau from SPAM Transgenic Mice Exhibit Potent Strain-Specific Prion-Like Seeding Properties Characteristic of Human Neurodegenerative DiseasesEthan D Smith, Giavanna Paterno, Brach M Bell, et al.
The Journal of Biological Chemistry|December 21, 2017
Distinct differences in prion-like seeding and aggregation between Tau protein variants provide mechanistic insights into tauopathiesKevin H Strang, Cara L Croft, Zachary A Sorrentino, et al.
Molecular Cell|June 3, 2014
A cellular system that degrades misfolded proteins and protects against neurodegenerationLili Guo, Benoit I Giasson, Alex Glavis-Bloom, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|November 14, 2008
Clinical and pathological characteristics of patients with leucine-rich repeat kinase-2 mutationsJason P Covy, Wuxing Yuan, Elisa A Waxman, et al.
The Journal of Cell Biology|May 5, 2004
Degradative organelles containing mislocalized alpha-and beta-synuclein proliferate in presenilin-1 null neuronsChristina A Wilson, Diane D Murphy, Benoit I Giasson, et al.
Plos One|April 3, 2018
Novel monoclonal antibodies targeting the microtubule-binding domain of human tauCara L Croft, Brenda D Moore, Yong Ran, et al.
Molecular and Cellular Neurosciences|June 4, 2018
Partial loss of ATP13A2 causes selective gliosis independent of robust lipofuscinosisSruti Rayaprolu, Yasin B Seven, John Howard, et al.
Pageof 18