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Advanced Science (Weinheim, Baden-Wurttemberg, Germany)|November 10, 2023
Spatial Mechano-Signaling Regulation of GTPases through Non-Degradative UbiquitinationRaj N Sewduth, Paolo Carai, Tonci Ivanisevic, et al.Circulation Research|March 17, 2020
The Noonan Syndrome Gene Lztr1 Controls Cardiovascular Function by Regulating Vesicular TraffickingRaj Nayan Sewduth, Silvia Pandolfi, Mikhail Steklov, et al.The EMBO Journal|June 10, 2024
K128 ubiquitination constrains RAS activity by expanding its binding interface with GAP proteinsWout Magits, Mikhail Steklov, Hyunbum Jang, et al.Molecular Neurobiology|April 21, 2019
LRP1 Has a Predominant Role in Production over Clearance of Aβ in a Mouse Model of Alzheimer's DiseaseBart Van Gool, Steffen E Storck, Sara M Reekmans, et al.Scientific Reports|February 19, 2021
Syntenin-knock out reduces exosome turnover and viral transductionRudra Kashyap, Marielle Balzano, Benoit Lechat, et al.Plos One|October 2, 2009
AAV-tau mediates pyramidal neurodegeneration by cell-cycle re-entry without neurofibrillary tangle formation in wild-type miceTomasz Jaworski, Ilse Dewachter, Benoit Lechat, et al.Nature|December 14, 2011
GSK-3α/β kinases and amyloid production in vivoTomasz Jaworski, Ilse Dewachter, Benoit Lechat, et al.Nature Communications|December 15, 2025
Intrinsic resistance to RAS inhibitors is driven by dysregulation of KRAS degradationTonci Ivanisevic, Yan Ma, Emiel Van Boxel, et al.Synapse (New York, N.Y.)|February 8, 2013
Glycogen synthase kinase-3β (GSK3β) expression in a mouse model of Alzheimer's disease: a light and electron microscopy studyJohanna C Gandy, Miguel Melendez-Ferro, Gautam N Bijur, et al.Plos One|February 6, 2014
Early structural and functional defects in synapses and myelinated axons in stratum lacunosum moleculare in two preclinical models for tauopathyHervé Maurin, Seon-Ah Chong, Igor Kraev, et al.Pageof 3