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Elife|August 3, 2021
EPHX1 mutations cause a lipoatrophic diabetes syndrome due to impaired epoxide hydrolysis and increased cellular senescenceJeremie Gautheron, Christophe Morisseau, Wendy K Chung, et al.
Hepatology (Baltimore, Md.)|April 18, 2018
The adenosine monophosphate-activated protein kinase-vacuolar adenosine triphosphatase-pH axis: A key regulator of the profibrogenic phenotype of human hepatic stellate cellsGiusi Marrone, Francesco De Chiara, Katrin Böttcher, et al.
Annals of the New York Academy of Sciences|October 23, 2009
Ion channel regulation by AMPK: the route of hypoxia-response coupling in thecarotid body and pulmonary arteryA Mark Evans, D Grahame Hardie, Chris Peers, et al.
Diabetes|April 28, 2005
Essential role of chicken ovalbumin upstream promoter-transcription factor II in insulin secretion and insulin sensitivity revealed by conditional gene knockoutPascale Bardoux, Pili Zhang, Daisy Flamez, et al.
Journal of Molecular and Cellular Cardiology|May 9, 2014
Reduced scar maturation and contractility lead to exaggerated left ventricular dilation after myocardial infarction in mice lacking AMPKα1Gauthier Noppe, Cécile Dufeys, Patricia Buchlin, et al.
Molecular Genetics and Metabolism|September 8, 2015
Biotin deprivation impairs mitochondrial structure and function and has implications for inherited metabolic disordersEstefanía Ochoa-Ruiz, Rodrigo Díaz-Ruiz, Alaín de J Hernández-Vázquez, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|October 20, 2021
Inactivation of AMPK Leads to Attenuation of Antigen Presentation and Immune Evasion in Lung AdenocarcinomaYajing Gao, Pekka Päivinen, Sushil Tripathi, et al.
Nature Medicine|March 24, 2015
Activation of AMPKα2 in adipocytes is essential for nicotine-induced insulin resistance in vivoYue Wu, Ping Song, Wencheng Zhang, et al.
Biochemical Pharmacology|December 17, 2014
Apoptolidins A and C activate AMPK in metabolically sensitive cell types and are mechanistically distinct from oligomycin AJeffrey D Serrill, Michelle Tan, Serge Fotso, et al.
Cardiovascular Research|July 9, 2018
α1AMPK deletion in myelomonocytic cells induces a pro-inflammatory phenotype and enhances angiotensin II-induced vascular dysfunctionThomas Jansen, Swenja Kröller-Schön, Tanja Schönfelder, et al.
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