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FEBS Letters|December 22, 2018
Mg2+ -free ATP regulates the processivity of native cytoplasmic dyneinVincent A Behrens, Wilhelm J Walter, Carsten Peters, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 20, 2014
ATP turnover by individual myosin molecules hints at two conformers of the myosin active siteMamta Amrute-Nayak, Katharina-Antonia Lambeck, Ante Radocaj, et al.
The Science of the Total Environment|October 4, 2016
Detection of Legionella-contaminated aerosols in the vicinity of a bio-trickling filter of a breeding sow facility - A pilot studySandra M Walser, Bernhard Brenner, Anika Wunderlich, et al.
Frontiers in Physiology|February 7, 2018
Differences in Contractile Function of Myofibrils within Human Embryonic Stem Cell-Derived Cardiomyocytes vs. Adult Ventricular Myofibrils Are Related to Distinct Sarcomeric Protein IsoformsBogdan Iorga, Kristin Schwanke, Natalie Weber, et al.
Frontiers in Physiology|April 9, 2020
Cycling Cross-Bridges Contribute to Thin Filament Activation in Human Slow-Twitch FibersAlfredo Jesus López-Dávila, Joseph M Chalovich, Stefan Zittrich, et al.
Physiological Genomics|May 22, 2003
Familial hypertrophic cardiomyopathy mutations in troponin I (K183D, G203S, K206Q) enhance filament slidingJan Köhler, Ying Chen, Bernhard Brenner, et al.
Nature Structural & Molecular Biology|January 6, 2009
The mechanism of pentabromopseudilin inhibition of myosin motor activityRoman Fedorov, Markus Böhl, Georgios Tsiavaliaris, et al.
Basic Research in Cardiology|July 20, 2011
Unequal allelic expression of wild-type and mutated β-myosin in familial hypertrophic cardiomyopathySnigdha Tripathi, Imke Schultz, Edgar Becker, et al.
Journal of Molecular and Cellular Cardiology|January 16, 2013
Familial hypertrophic cardiomyopathy: functional effects of myosin mutation R723G in cardiomyocytesTheresia Kraft, E Rosalie Witjas-Paalberends, Nicky M Boontje, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 20, 2004
Hypertrophic cardiomyopathy-related beta-myosin mutations cause highly variable calcium sensitivity with functional imbalances among individual muscle cellsSebastian E Kirschner, Edgar Becker, Massimo Antognozzi, et al.
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