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The Journal of Clinical Investigation|June 1, 2022
MicroRNAs as therapeutic targets in cardiovascular diseaseBernhard Laggerbauer, Stefan Engelhardt
Trends in Molecular Medicine|February 14, 2006
Spinal muscular atrophy: the RNP connectionChristian Eggert, Ashwin Chari, Bernhard Laggerbauer, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|August 23, 2016
Viral Vector-Based Targeting of miR-21 in Cardiac Nonmyocyte Cells Reduces Pathologic Remodeling of the HeartDeepak Ramanujam, Yassine Sassi, Bernhard Laggerbauer, et al.
RNA Biology|May 23, 2013
The human transcriptome is enriched for miRNA-binding sites located in cooperativity-permitting distanceAndrea Rinck, Martin Preusse, Bernhard Laggerbauer, et al.
Genes & Development|October 6, 2005
Reduced U snRNP assembly causes motor axon degeneration in an animal model for spinal muscular atrophyChristoph Winkler, Christian Eggert, Dietmar Gradl, et al.
Human Molecular Genetics|July 31, 2008
Tdrd3 is a novel stress granule-associated protein interacting with the Fragile-X syndrome protein FMRPBastian Linder, Oliver Plöttner, Matthias Kroiss, et al.
Journal of Molecular and Cellular Cardiology|August 2, 2011
A phenotypic screen to identify hypertrophy-modulating microRNAs in primary cardiomyocytesClaudia Jentzsch, Simon Leierseder, Xavier Loyer, et al.
Plos One|November 11, 2014
Identification of a PRPF4 loss-of-function variant that abrogates U4/U6.U5 tri-snRNP integration and is associated with retinitis pigmentosaBastian Linder, Anja Hirmer, Andreas Gal, et al.
RNA (New York, N.Y.)|April 21, 2005
The human U5 snRNP 52K protein (CD2BP2) interacts with U5-102K (hPrp6), a U4/U6.U5 tri-snRNP bridging protein, but dissociates upon tri-snRNP formationBernhard Laggerbauer, Sunbin Liu, Evgeny Makarov, et al.
Human Molecular Genetics|November 6, 2010
Systemic splicing factor deficiency causes tissue-specific defects: a zebrafish model for retinitis pigmentosaBastian Linder, Holger Dill, Anja Hirmer, et al.
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