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Kidney & Blood Pressure Research
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January 16, 2024
SGLT2 Inhibitors Decrease Overhydration and Proteasuria in Patients with Chronic Kidney Disease: A Longitudinal Observational Study
Anja Schork, Marie-Luise Eberbach, Bernhard N Bohnert, et al.
Transplant International : Official Journal of the European Society for Organ Transplantation
|
June 25, 2024
SGLT2 Inhibitors Correct Fluid Overload in Adult Kidney Transplant Recipients-A Prospective Observational Study
Anja Schork, Marie-Luise Eberbach, Ferruh Artunc, et al.
Pflugers Archiv : European Journal of Physiology
|
January 3, 2026
Activation of the alternative complement pathway and its relevance for sodium retention in experimental nephrotic syndrome
Daniel Essigke, M Zaher Kalo, Lingsi Kong, et al.
Kidney International Reports
|
April 14, 2025
GFR is a Key Determinant of Red Blood Cell Survival in Anemia Associated With Progressive CKD
Rosi Bissinger, Lina Schaefer, Bernhard N Bohnert, et al.
Research Square
|
September 5, 2025
Activation of the alternative complement pathway and its relevance for sodium retention in experimental nephrotic syndrome
Daniel Essigke, M Zaher Kalo, Lingsi Kong, et al.
Acta Physiologica (Oxford, England)
|
March 2, 2021
Zymogen-locked mutant prostasin (Prss8) leads to incomplete proteolytic activation of the epithelial sodium channel (ENaC) and severely compromises triamterene tolerance in mice
Daniel Essigke, Alexandr V Ilyaskin, Matthias Wörn, et al.
American Journal of Physiology. Renal Physiology
|
August 23, 2021
Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na<sup>+</sup> channel in the mouse kidney
Bernhard N Bohnert, Daniel Essigke, Andrea Janessa, et al.
Acta Physiologica (Oxford, England)
|
April 22, 2019
Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome
Bernhard N Bohnert, Sophie Daiminger, Matthias Wörn, et al.
Kidney International
|
October 19, 2017
Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome
Bernhard N Bohnert, Martina Menacher, Andrea Janessa, et al.
Acta Physiologica (Oxford, England)
|
May 27, 2020
Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome
Mengyun Xiao, Bernhard N Bohnert, Hande Aypek, et al.
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Search research articles
Search
Showing results (21-30 of 33) with videos related to
Sort By:
Page
of 4
Kidney & Blood Pressure Research
|
January 16, 2024
SGLT2 Inhibitors Decrease Overhydration and Proteasuria in Patients with Chronic Kidney Disease: A Longitudinal Observational Study
Anja Schork, Marie-Luise Eberbach, Bernhard N Bohnert, et al.
Transplant International : Official Journal of the European Society for Organ Transplantation
|
June 25, 2024
SGLT2 Inhibitors Correct Fluid Overload in Adult Kidney Transplant Recipients-A Prospective Observational Study
Anja Schork, Marie-Luise Eberbach, Ferruh Artunc, et al.
Pflugers Archiv : European Journal of Physiology
|
January 3, 2026
Activation of the alternative complement pathway and its relevance for sodium retention in experimental nephrotic syndrome
Daniel Essigke, M Zaher Kalo, Lingsi Kong, et al.
Kidney International Reports
|
April 14, 2025
GFR is a Key Determinant of Red Blood Cell Survival in Anemia Associated With Progressive CKD
Rosi Bissinger, Lina Schaefer, Bernhard N Bohnert, et al.
Research Square
|
September 5, 2025
Activation of the alternative complement pathway and its relevance for sodium retention in experimental nephrotic syndrome
Daniel Essigke, M Zaher Kalo, Lingsi Kong, et al.
Acta Physiologica (Oxford, England)
|
March 2, 2021
Zymogen-locked mutant prostasin (Prss8) leads to incomplete proteolytic activation of the epithelial sodium channel (ENaC) and severely compromises triamterene tolerance in mice
Daniel Essigke, Alexandr V Ilyaskin, Matthias Wörn, et al.
American Journal of Physiology. Renal Physiology
|
August 23, 2021
Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na<sup>+</sup> channel in the mouse kidney
Bernhard N Bohnert, Daniel Essigke, Andrea Janessa, et al.
Acta Physiologica (Oxford, England)
|
April 22, 2019
Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome
Bernhard N Bohnert, Sophie Daiminger, Matthias Wörn, et al.
Kidney International
|
October 19, 2017
Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome
Bernhard N Bohnert, Martina Menacher, Andrea Janessa, et al.
Acta Physiologica (Oxford, England)
|
May 27, 2020
Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome
Mengyun Xiao, Bernhard N Bohnert, Hande Aypek, et al.
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of 4