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Bethany W Long

Showing results (1-10 of 11) with videos related to

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Frontiers in Cell and Developmental Biology|August 19, 2016
A New Method to Stabilize C-Kit Expression in Reparative Cardiac Mesenchymal CellsMarcin Wysoczynski, Sujith Dassanayaka, Ayesha Zafir, et al.
Plos One|November 14, 2015
O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal CellsAyesha Zafir, James A Bradley, Bethany W Long, et al.
The Biochemical Journal|January 29, 2015
High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylationSujith Dassanayaka, Ryan D Readnower, Joshua K Salabei, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 30, 2010
O-linked β-N-acetylglucosamine transferase is indispensable in the failing heartLewis J Watson, Heberty T Facundo, Gladys A Ngoh, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 5, 2013
Cardiomyocyte Ogt is essential for postnatal viabilityLewis J Watson, Bethany W Long, Angelica M DeMartino, et al.
Stem Cells (Dayton, Ohio)|January 22, 2013
Protein O-GlcNAcylation is a novel cytoprotective signal in cardiac stem cellsAyesha Zafir, Ryan Readnower, Bethany W Long, et al.
Redox Biology|June 10, 2018
Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overloadSujith Dassanayaka, Yuting Zheng, Andrew A Gibb, et al.
Basic Research in Cardiology|June 2, 2019
E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylationSujith Dassanayaka, Kenneth R Brittian, Andrea Jurkovic, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 23, 2020
Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarctionTimothy N Audam, Yibing Nong, Alex Tomlin, et al.
Basic Research in Cardiology|March 17, 2017
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Frontiers in Cell and Developmental Biology|August 19, 2016
A New Method to Stabilize C-Kit Expression in Reparative Cardiac Mesenchymal CellsMarcin Wysoczynski, Sujith Dassanayaka, Ayesha Zafir, et al.
Plos One|November 14, 2015
O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal CellsAyesha Zafir, James A Bradley, Bethany W Long, et al.
The Biochemical Journal|January 29, 2015
High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylationSujith Dassanayaka, Ryan D Readnower, Joshua K Salabei, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 30, 2010
O-linked β-N-acetylglucosamine transferase is indispensable in the failing heartLewis J Watson, Heberty T Facundo, Gladys A Ngoh, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 5, 2013
Cardiomyocyte Ogt is essential for postnatal viabilityLewis J Watson, Bethany W Long, Angelica M DeMartino, et al.
Stem Cells (Dayton, Ohio)|January 22, 2013
Protein O-GlcNAcylation is a novel cytoprotective signal in cardiac stem cellsAyesha Zafir, Ryan Readnower, Bethany W Long, et al.
Redox Biology|June 10, 2018
Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overloadSujith Dassanayaka, Yuting Zheng, Andrew A Gibb, et al.
Basic Research in Cardiology|June 2, 2019
E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylationSujith Dassanayaka, Kenneth R Brittian, Andrea Jurkovic, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 23, 2020
Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarctionTimothy N Audam, Yibing Nong, Alex Tomlin, et al.
Basic Research in Cardiology|March 17, 2017
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.
Pageof 2