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Betty M Tijms

Showing results (1-10 of 167) with videos related to

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Brain : a Journal of Neurology|September 28, 2019
Elevated tau PET signal depends on abnormal amyloid levels and is uncommon in unimpaired individualsBrian A Gordon, Betty M Tijms
Brain : a Journal of Neurology|February 12, 2021
Concatenating plasma p-tau to Alzheimer's diseaseBetty M Tijms, Charlotte E Teunissen
Brain : a Journal of Neurology|May 26, 2018
Chasing the start of sporadic Alzheimer's disease running in familiesBetty M Tijms, Pieter Jelle Visser
The Lancet. Neurology|February 5, 2018
Capturing the Alzheimer's disease pathological cascadeBetty M Tijms, Pieter Jelle Visser
Nature Reviews. Neurology|September 1, 2023
Treatments for AD: towards the right target at the right timeWiesje M van der Flier, Betty M Tijms
Neurobiology of Aging|August 10, 2021
Proteomic correlates of cortical thickness in cognitively normal individuals with normal and abnormal cerebrospinal fluid beta-amyloid<sub>1-42</sub>Laura L Ekblad, Pieter Jelle Visser, Betty M Tijms, et al.
Cerebral Cortex (New York, N.Y. : 1991)|September 1, 2011
Similarity-based extraction of individual networks from gray matter MRI scansBetty M Tijms, Peggy Seriès, David J Willshaw, et al.
Alzheimer'S Research & Therapy|February 15, 2017
Unbiased estimates of cerebrospinal fluid β-amyloid 1-42 cutoffs in a large memory clinic populationDaniela Bertens, Betty M Tijms, Philip Scheltens, et al.
Critical Reviews in Clinical Laboratory Sciences|November 8, 2019
Cerebrospinal fluid proteomics and biological heterogeneity in Alzheimer's disease: A literature reviewKirsten E J Wesenhagen, Charlotte E Teunissen, Pieter Jelle Visser, et al.
Neurobiology of Aging|June 26, 2020
Single-subject gray matter networks predict future cortical atrophy in preclinical Alzheimer's diseaseEllen Dicks, Wiesje M van der Flier, Philip Scheltens, et al.
Pageof 17

Showing results (1-10 of 167) with videos related to

Sort By:
Pageof 17
Brain : a Journal of Neurology|September 28, 2019
Elevated tau PET signal depends on abnormal amyloid levels and is uncommon in unimpaired individualsBrian A Gordon, Betty M Tijms
Brain : a Journal of Neurology|February 12, 2021
Concatenating plasma p-tau to Alzheimer's diseaseBetty M Tijms, Charlotte E Teunissen
Brain : a Journal of Neurology|May 26, 2018
Chasing the start of sporadic Alzheimer's disease running in familiesBetty M Tijms, Pieter Jelle Visser
The Lancet. Neurology|February 5, 2018
Capturing the Alzheimer's disease pathological cascadeBetty M Tijms, Pieter Jelle Visser
Nature Reviews. Neurology|September 1, 2023
Treatments for AD: towards the right target at the right timeWiesje M van der Flier, Betty M Tijms
Neurobiology of Aging|August 10, 2021
Proteomic correlates of cortical thickness in cognitively normal individuals with normal and abnormal cerebrospinal fluid beta-amyloid<sub>1-42</sub>Laura L Ekblad, Pieter Jelle Visser, Betty M Tijms, et al.
Cerebral Cortex (New York, N.Y. : 1991)|September 1, 2011
Similarity-based extraction of individual networks from gray matter MRI scansBetty M Tijms, Peggy Seriès, David J Willshaw, et al.
Alzheimer'S Research & Therapy|February 15, 2017
Unbiased estimates of cerebrospinal fluid β-amyloid 1-42 cutoffs in a large memory clinic populationDaniela Bertens, Betty M Tijms, Philip Scheltens, et al.
Critical Reviews in Clinical Laboratory Sciences|November 8, 2019
Cerebrospinal fluid proteomics and biological heterogeneity in Alzheimer's disease: A literature reviewKirsten E J Wesenhagen, Charlotte E Teunissen, Pieter Jelle Visser, et al.
Neurobiology of Aging|June 26, 2020
Single-subject gray matter networks predict future cortical atrophy in preclinical Alzheimer's diseaseEllen Dicks, Wiesje M van der Flier, Philip Scheltens, et al.
Pageof 17