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Journal of Investigative Medicine : the Official Publication of the American Federation for Clinical Research|December 16, 2005
Guanosine triphosphatase activation occurs downstream of calcineurin in cardiac hypertrophy*Kenneth E Richardson, Paul Tannous, Kambeez Berenji, et al.
Journal of Bioenergetics and Biomembranes|January 25, 2011
The complex interplay between mitochondrial dynamics and cardiac metabolismValentina Parra, Hugo Verdejo, Andrea del Campo, et al.
Journal of Molecular and Cellular Cardiology|November 24, 2011
STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophyXiang Luo, Berdymammet Hojayev, Nan Jiang, et al.
Endocrine Reviews|March 18, 2020
Is Mitochondrial Dysfunction a Common Root of Noncommunicable Chronic Diseases?Alexis Diaz-Vegas, Pablo Sanchez-Aguilera, James R Krycer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 30, 2007
The Down syndrome critical region protein RCAN1 regulates long-term potentiation and memory via inhibition of phosphatase signalingCharles A Hoeffer, Asim Dey, Nita Sachan, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 8, 2003
Dual roles of modulatory calcineurin-interacting protein 1 in cardiac hypertrophyRick B Vega, Beverly A Rothermel, Carla J Weinheimer, et al.
Circulation Research|May 30, 2009
Physical and functional interaction between calcineurin and the cardiac L-type Ca2+ channelSamvit Tandan, Yanggan Wang, Thomas T Wang, et al.
Circulation|June 11, 2008
Intracellular protein aggregation is a proximal trigger of cardiomyocyte autophagyPaul Tannous, Hongxin Zhu, Andriy Nemchenko, et al.
The Journal of Clinical Investigation|July 4, 2007
Cardiac autophagy is a maladaptive response to hemodynamic stressHongxin Zhu, Paul Tannous, Janet L Johnstone, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 4, 2011
Histone deacetylase (HDAC) inhibitors attenuate cardiac hypertrophy by suppressing autophagyDian J Cao, Zhao V Wang, Pavan K Battiprolu, et al.
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