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Beverly L Davidson

Showing results (121-130 of 205) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|June 5, 2003
Allele-specific silencing of dominant disease genesVictor M Miller, Haibin Xia, Ginger L Marrs, et al.
Human Molecular Genetics|September 22, 2018
The long non-coding RNA NEAT1 is elevated in polyglutamine repeat expansion diseases and protects from disease gene-dependent toxicitiesCongsheng Cheng, Ryan M Spengler, Megan S Keiser, et al.
Stem Cell Research|January 6, 2019
Generation of Spinocerebellar Ataxia Type 2 induced pluripotent stem cell lines, CHOPi002-A and CHOPi003-A, from patients with abnormal CAG repeats in the coding region of the ATXN2 geneJean Ann Maguire, Alyssa L Gagne, Pedro Gonzalez-Alegre, et al.
Journal of Virology|September 16, 2006
Optimization of feline immunodeficiency virus vectors for RNA interferenceScott Q Harper, Patrick D Staber, Christine R Beck, et al.
Neuron|May 6, 2016
PIAS1 Regulates Mutant Huntingtin Accumulation and Huntington's Disease-Associated Phenotypes In VivoJoseph Ochaba, Alex Mas Monteys, Jacqueline G O'Rourke, et al.
Biorxiv : the Preprint Server for Biology|September 4, 2024
Generation of humanized mouse models to support therapeutic development for SYNGAP1 and STXBP1 disordersAlex J Felix, Taryn Wilson, Rani Randell, et al.
Neuron|January 6, 2015
Reinstating aberrant mTORC1 activity in Huntington's disease mice improves disease phenotypesJohn H Lee, Luis Tecedor, Yong Hong Chen, et al.
JCI Insight|October 2, 2019
Neuronal network dysfunction precedes storage and neurodegeneration in a lysosomal storage disorderRebecca C Ahrens-Nicklas, Luis Tecedor, Arron F Hall, et al.
Journal of Huntington'S Disease|July 27, 2014
Rhes suppression enhances disease phenotypes in Huntington's disease miceJohn H Lee, Matthew J Sowada, Ryan L Boudreau, et al.
Plos One|August 29, 2022
Huntington's disease phenotypes are improved via mTORC1 modulation by small molecule therapySophie St-Cyr, Daniel D Child, Emilie Giaime, et al.
Pageof 21

Showing results (121-130 of 205) with videos related to

Sort By:
Pageof 21
Proceedings of the National Academy of Sciences of the United States of America|June 5, 2003
Allele-specific silencing of dominant disease genesVictor M Miller, Haibin Xia, Ginger L Marrs, et al.
Human Molecular Genetics|September 22, 2018
The long non-coding RNA NEAT1 is elevated in polyglutamine repeat expansion diseases and protects from disease gene-dependent toxicitiesCongsheng Cheng, Ryan M Spengler, Megan S Keiser, et al.
Stem Cell Research|January 6, 2019
Generation of Spinocerebellar Ataxia Type 2 induced pluripotent stem cell lines, CHOPi002-A and CHOPi003-A, from patients with abnormal CAG repeats in the coding region of the ATXN2 geneJean Ann Maguire, Alyssa L Gagne, Pedro Gonzalez-Alegre, et al.
Journal of Virology|September 16, 2006
Optimization of feline immunodeficiency virus vectors for RNA interferenceScott Q Harper, Patrick D Staber, Christine R Beck, et al.
Neuron|May 6, 2016
PIAS1 Regulates Mutant Huntingtin Accumulation and Huntington's Disease-Associated Phenotypes In VivoJoseph Ochaba, Alex Mas Monteys, Jacqueline G O'Rourke, et al.
Biorxiv : the Preprint Server for Biology|September 4, 2024
Generation of humanized mouse models to support therapeutic development for SYNGAP1 and STXBP1 disordersAlex J Felix, Taryn Wilson, Rani Randell, et al.
Neuron|January 6, 2015
Reinstating aberrant mTORC1 activity in Huntington's disease mice improves disease phenotypesJohn H Lee, Luis Tecedor, Yong Hong Chen, et al.
JCI Insight|October 2, 2019
Neuronal network dysfunction precedes storage and neurodegeneration in a lysosomal storage disorderRebecca C Ahrens-Nicklas, Luis Tecedor, Arron F Hall, et al.
Journal of Huntington'S Disease|July 27, 2014
Rhes suppression enhances disease phenotypes in Huntington's disease miceJohn H Lee, Matthew J Sowada, Ryan L Boudreau, et al.
Plos One|August 29, 2022
Huntington's disease phenotypes are improved via mTORC1 modulation by small molecule therapySophie St-Cyr, Daniel D Child, Emilie Giaime, et al.
Pageof 21