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Antioxidants & Redox Signaling|April 2, 2015
Do Beta 2-Glycoprotein I Disulfide Bonds Protect the Human Retina in the Setting of Age-Related Macular Degeneration?Miao Qi, Mahmoud Abdelatti, Matthew Krilis, et al.
Plos One|April 1, 2016
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion InjuryPeng Zhang, James C Weaver, Gang Chen, et al.
Scientific Reports|September 28, 2016
Gram Negative Bacterial Inflammation Ameliorated by the Plasma Protein Beta 2-Glycoprotein ISaijun Zhou, Gang Chen, Miao Qi, et al.
Blood|June 17, 2010
Naturally occurring free thiols within beta 2-glycoprotein I in vivo: nitrosylation, redox modification by endothelial cells, and regulation of oxidative stress-induced cell injuryYiannis Ioannou, Jing-Yun Zhang, Freda H Passam, et al.
Arthritis and Rheumatism|July 27, 2006
Anti-beta2-glycoprotein I antibodies in complex with beta2-glycoprotein I can activate platelets in a dysregulated manner via glycoprotein Ib-IX-VTong Shi, Bill Giannakopoulos, Xiaokai Yan, et al.
Free Radical Biology & Medicine|September 26, 2018
Dual roles of different redox forms of complement factor H in protecting against age related macular degenerationMatthew Krilis, Miao Qi, Jian Qi, et al.
Journal of Autoimmunity|June 19, 2012
Factor XI is a substrate for oxidoreductases: enhanced activation of reduced FXI and its role in antiphospholipid syndrome thrombosisBill Giannakopoulos, Lu Gao, Miao Qi, et al.
Oncotarget|February 5, 2017
Nitration of tyrosines in complement factor H domains alters its immunological activity and mediates a pathogenic role in age related macular degenerationMatthew Krilis, Miao Qi, Michele C Madigan, et al.
Plos One|March 17, 2016
CD117+ Dendritic and Mast Cells Are Dependent on RasGRP4 to Function as Accessory Cells for Optimal Natural Killer Cell-Mediated Responses to LipopolysaccharideSaijun Zhou, Kumiko Tanaka, Meredith O'Keeffe, et al.
Arthritis and Rheumatism|May 28, 2011
Novel assays of thrombogenic pathogenicity in the antiphospholipid syndrome based on the detection of molecular oxidative modification of the major autoantigen β2-glycoprotein IYiannis Ioannou, Jing-Yun Zhang, Miao Qi, et al.
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