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Journal of Atherosclerosis and Thrombosis|February 4, 2010
Adipose tissue, inflammation and atherosclerosisBirgit Gustafson
The Journal of Biological Chemistry|February 25, 2010
Activation of canonical wingless-type MMTV integration site family (Wnt) signaling in mature adipocytes increases beta-catenin levels and leads to cell dedifferentiation and insulin resistanceBirgit Gustafson, Ulf Smith
Atherosclerosis|May 11, 2015
Regulation of white adipogenesis and its relation to ectopic fat accumulation and cardiovascular riskBirgit Gustafson, Ulf Smith
Diabetes|March 27, 2012
The WNT inhibitor Dickkopf 1 and bone morphogenetic protein 4 rescue adipogenesis in hypertrophic obesity in humansBirgit Gustafson, Ulf Smith
The Journal of Biological Chemistry|February 9, 2006
Cytokines promote Wnt signaling and inflammation and impair the normal differentiation and lipid accumulation in 3T3-L1 preadipocytesBirgit Gustafson, Ulf Smith
Nature Communications|June 23, 2019
Reduced subcutaneous adipogenesis in human hypertrophic obesity is linked to senescent precursor cellsBirgit Gustafson, Annika Nerstedt, Ulf Smith
Biochemical and Biophysical Research Communications|April 20, 2007
Wnt-signaling is maintained and adipogenesis inhibited by TNFalpha but not MCP-1 and resistinAnn Hammarstedt, Petter Isakson, Birgit Gustafson, et al.
Diabetes|April 9, 2009
Impaired preadipocyte differentiation in human abdominal obesity: role of Wnt, tumor necrosis factor-alpha, and inflammationPetter Isakson, Ann Hammarstedt, Birgit Gustafson, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|September 8, 2007
Inflamed adipose tissue: a culprit underlying the metabolic syndrome and atherosclerosisBirgit Gustafson, Ann Hammarstedt, Christian X Andersson, et al.
Biochemical and Biophysical Research Communications|August 21, 2003
Adiponectin gene activation by thiazolidinediones requires PPAR gamma 2, but not C/EBP alpha-evidence for differential regulation of the aP2 and adiponectin genesBirgit Gustafson, Maia M Jack, Samuel W Cushman, et al.
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