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ACS Medicinal Chemistry Letters|November 17, 2022
Structure-Activity Relationships for the N-Me- Versus N-H-Amide Modification to Macrocyclic ent-Verticilide AntiarrhythmicsAbigail N Smith, Madelaine P Thorpe, Daniel J Blackwell, et al.Circulation Research|December 10, 2020
RYR2 Channel Inhibition Is the Principal Mechanism of Flecainide Action in CPVTDmytro O Kryshtal, Daniel J Blackwell, Christian L Egly, et al.Medrxiv : the Preprint Server for Health Sciences|April 1, 2025
Structural Evaluation of RYR2-CPVT Missense Variants and Continuous Bayesian Estimates of their PenetranceKohei Yamauchi, Matthew Ku, Devyn W Mitchell, et al.The Journal of Pharmacology and Experimental Therapeutics|March 9, 2023
In Vivo Pharmacokinetic and Pharmacodynamic Properties of the Antiarrhythmic Molecule ent-VerticilideDaniel J Blackwell, Abigail N Smith, Tri Do, et al.Journal of Molecular and Cellular Cardiology Plus|May 9, 2023
Low expression of the K280N TNNT2 mutation is sufficient to increase basal myofilament activation in human hypertrophy cardiomyopathyVasco Sequeira, Lili Wang, Paul J M Wijnker, et al.Journal of Molecular and Cellular Cardiology Plus|December 18, 2024
Conditional ablation of MCU exacerbated cardiac pathology in a genetic arrhythmic model of CPVTArpita Deb, Brian D Tow, Jie Hao, et al.Iscience|April 14, 2020
MCL-1 Inhibition by Selective BH3 Mimetics Disrupts Mitochondrial Dynamics Causing Loss of Viability and Functionality of Human CardiomyocytesMegan L Rasmussen, Nilay Taneja, Abigail C Neininger, et al.Proceedings of the National Academy of Sciences of the United States of America|February 23, 2019
Unnatural verticilide enantiomer inhibits type 2 ryanodine receptor-mediated calcium leak and is antiarrhythmicSuzanne M Batiste, Daniel J Blackwell, Kyungsoo Kim, et al.Structure (London, England : 1993)|April 3, 2018
A Mechanism of Calmodulin Modulation of the Human Cardiac Sodium ChannelChristopher N Johnson, Franck Potet, Matthew K Thompson, et al.Heart Rhythm|April 21, 2019
Patient-independent human induced pluripotent stem cell model: A new tool for rapid determination of genetic variant pathogenicity in long QT syndromeNikhil V Chavali, Dmytro O Kryshtal, Shan S Parikh, et al.Pageof 8