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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|November 24, 2020
Crosstalk between head and neck cancer cells and lymphatic endothelial cells promotes tumor metastasis via CXCL5-CXCR2 signalingBok-Soon Lee, Jeon Yeob Jang, Chorong Seo, et al.Cellular Signalling|December 17, 2008
Hepatocyte growth factor induces delayed STAT3 phosphorylation through interleukin-6 expressionBok-Soon Lee, Minseon Park, Hyun-Young Cha, et al.Research Square|July 18, 2023
Expression analysis of BACH1 with clinical variables using the US breast cancer patient cohortN Maritza Dowling, Galina Khramtsova, Olufunmilayo Olopade, et al.International Journal of Molecular Sciences|March 13, 2025
Tumor Regulatory Effect of 15-Hydroxyprostaglandin Dehydrogenase (HPGD) in Triple-Negative Breast CancerJoselyn Padilla, Bok-Soon Lee, Allen Kim, et al.Current Oncology (Toronto, Ont.)|July 25, 2025
Differential BACH1 Expression in Basal-like Breast Tumors of Black Women Identified via ImmunohistochemistryN M Dowling, Galina Khramtsova, Olufunmilayo Olopade, et al.Experimental & Molecular Medicine|September 15, 2004
Transforming variant of Met receptor confers serum independence and anti-apoptotic property and could be involved in the mouse thymic lymphomagenesisCheol-Min Baek, Soung-Hoo Jeon, Ja-June Jang, et al.Endocrine-Related Cancer|February 23, 2013
AY4, an agonistic anti-death receptor 4 MAB, induces apoptotic cell death in anaplastic thyroid cancer cells via downregulation of Bcl-xL with reactive oxygen species generationBok-Soon Lee, Hyun-Young Cha, Yoo Seob Shin, et al.Molecular Carcinogenesis|January 25, 2019
Identification of epithelial-specific ETS-1 (ESE-1) as a tumor suppressor and molecular target of green tea compound, EGCGTaekyu Ha, Jihye Lee, Zhiyuan Lou, et al.Cells|April 12, 2022
A Heme-Binding Transcription Factor BACH1 Regulates Lactate Catabolism Suggesting a Combined Therapy for Triple-Negative Breast CancerJoselyn Padilla, Bok-Soon Lee, Karen Zhai, et al.Radiation Oncology Journal|January 13, 2016
Met inactivation by S-allylcysteine suppresses the migration and invasion of nasopharyngeal cancer cells induced by hepatocyte growth factorOyeon Cho, Hye-Sook Hwang, Bok-Soon Lee, et al.Pageof 4