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Acta Neuropathologica Communications|November 20, 2016
Transgenic mice overexpressing the ALS-linked protein Matrin 3 develop a profound muscle phenotypeChristina Moloney, Sruti Rayaprolu, John Howard, et al.
Human Molecular Genetics|March 26, 2002
Early phenotypes that presage late-onset neurodegenerative disease allow testing of modifiers in Hdh CAG knock-in miceVanessa C Wheeler, Claire-Anne Gutekunst, Vladimir Vrbanac, et al.
Acta Neuropathologica Communications|December 15, 2017
Retraction Note: Transgenic mice overexpressing the ALS-linked protein Matrin 3 develop a profound muscle phenotypeChristina Moloney, Sruti Rayaprolu, John Howard, et al.
Brain Research|June 19, 2013
Unbiased screen reveals ubiquilin-1 and -2 highly associated with huntingtin inclusionsNicola J Rutherford, Jada Lewis, Amy K Clippinger, et al.
Neurobiology of Disease|September 17, 2025
Comparison of the ApoE allelic variants in the formation of intracerebral Aβ depositsGuilian Xu, Conner Angelle, Divya Huilgol, et al.
Human Molecular Genetics|April 26, 2011
Transgenic mice expressing caspase-6-derived N-terminal fragments of mutant huntingtin develop neurologic abnormalities with predominant cytoplasmic inclusion pathology composed largely of a smaller proteolytic derivativeAndrew T N Tebbenkamp, Cameron Green, Guilian Xu, et al.
Human Molecular Genetics|August 8, 2002
Polyglutamine and transcription: gene expression changes shared by DRPLA and Huntington's disease mouse models reveal context-independent effectsRuth Luthi-Carter, Andrew D Strand, Sarah A Hanson, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 9, 2014
Intramuscular injection of α-synuclein induces CNS α-synuclein pathology and a rapid-onset motor phenotype in transgenic miceAmanda N Sacino, Mieu Brooks, Michael A Thomas, et al.
Neurobiology of Disease|July 10, 2001
Distinct behavioral and neuropathological abnormalities in transgenic mouse models of HD and DRPLAG Schilling, H A Jinnah, V Gonzales, et al.
Neuron|February 1, 1997
ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusionsL I Bruijn, M W Becher, M K Lee, et al.
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