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Plos Medicine|November 11, 2005
Persistent amyloidosis following suppression of Abeta production in a transgenic model of Alzheimer diseaseJoanna L Jankowsky, Hilda H Slunt, Victoria Gonzales, et al.Ciba Foundation Symposium|January 1, 1996
Motor neuron disease and model systems: aetiologies, mechanisms and therapiesD L Price, V E Koliatsos, P C Wong, et al.The Journal of Biological Chemistry|January 15, 2008
Detergent-insoluble aggregates associated with amyotrophic lateral sclerosis in transgenic mice contain primarily full-length, unmodified superoxide dismutase-1Bryan F Shaw, Herman L Lelie, Armando Durazo, et al.Angewandte Chemie (International Ed. in English)|April 20, 2021
Supercharging Prions via Amyloid-Selective Lysine AcetylationKatelyn M Baumer, Christopher D Cook, Collin T Zahler, et al.Neurobiology of Disease|September 10, 2005
Progressive phenotype and nuclear accumulation of an amino-terminal cleavage fragment in a transgenic mouse model with inducible expression of full-length mutant huntingtinYuji Tanaka, Shuichi Igarashi, Masayuki Nakamura, et al.Human Molecular Genetics|December 3, 2003
Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretaseJoanna L Jankowsky, Daniel J Fadale, Jeffrey Anderson, et al.Journal of Alzheimer'S Disease : JAD|January 7, 2017
Quantitative Comparison of Dense-Core Amyloid Plaque Accumulation in Amyloid-β Protein Precursor Transgenic MicePeng Liu, John H Reichl, Eshaan R Rao, et al.Neurobiology of Disease|July 13, 2001
Creatine increase survival and delays motor symptoms in a transgenic animal model of Huntington's diseaseO A Andreassen, A Dedeoglu, R J Ferrante, et al.Human Molecular Genetics|May 23, 1998
Truncated N-terminal fragments of huntingtin with expanded glutamine repeats form nuclear and cytoplasmic aggregates in cell cultureJ K Cooper, G Schilling, M F Peters, et al.Nature Medicine|July 1, 1997
Hyperaccumulation of FAD-linked presenilin 1 variants in vivoM K Lee, D R Borchelt, G Kim, et al.Pageof 28