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Proceedings of the National Academy of Sciences of the United States of America|July 12, 2005
The C-terminal lysines fine-tune P53 stress responses in a mouse model but are not required for stability control or transactivationKurt A Krummel, Crystal J Lee, Franck Toledo, et al.
Nucleic Acids Research|July 28, 2006
RMCE-ASAP: a gene targeting method for ES and somatic cells to accelerate phenotype analysesFranck Toledo, Chung-Wen Liu, Crystal J Lee, et al.
Disease Models & Mechanisms|September 4, 2023
A systematic approach identifies p53-DREAM pathway target genes associated with blood or brain abnormalitiesJeanne Rakotopare, Vincent Lejour, Carla Duval, et al.
Brain : a Journal of Neurology|March 19, 2009
Distinct phenotypic and functional features of CADASIL mutations in the Notch3 ligand binding domainMarie Monet-Leprêtre, Boris Bardot, Barbara Lemaire, et al.
Molecular and Cellular Biology|December 13, 2006
Mouse mutants reveal that putative protein interaction sites in the p53 proline-rich domain are dispensable for tumor suppressionFranck Toledo, Crystal J Lee, Kurt A Krummel, et al.
NPJ Genomic Medicine|April 18, 2026
Low-penetrance TP53 variants are mainly hypomorphic: an underestimated issue with high clinical significanceLea Rodriguez, Bernard Leroy, Franck Toledo, et al.
Genes & Development|March 31, 2017
Essential role for centromeric factors following p53 loss and oncogenic transformationDan Filipescu, Monica Naughtin, Katrina Podsypanina, et al.
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