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Boris Rogelj

Showing results (61-70 of 82) with videos related to

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Nature Neuroscience|March 2, 2011
Characterizing the RNA targets and position-dependent splicing regulation by TDP-43James R Tollervey, Tomaž Curk, Boris Rogelj, et al.
Journal of Cell Science|February 13, 2019
Nuclear RNA foci from <i>C9ORF72</i> expansion mutation form paraspeckle-like bodiesAna Bajc Česnik, Simona Darovic, Sonja Prpar Mihevc, et al.
Plos One|March 22, 2014
Allele-specific knockdown of ALS-associated mutant TDP-43 in neural stem cells derived from induced pluripotent stem cellsAgnes L Nishimura, Carole Shum, Emma L Scotter, et al.
Human Molecular Genetics|October 4, 2017
C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicityYoun-Bok Lee, Pranetha Baskaran, Jorge Gomez-Deza, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|December 21, 2011
An MND/ALS phenotype associated with C9orf72 repeat expansion: abundant p62-positive, TDP-43-negative inclusions in cerebral cortex, hippocampus and cerebellum but without associated cognitive declineClaire Troakes, Satomi Maekawa, Lokesh Wijesekera, et al.
Human Molecular Genetics|September 5, 2020
C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicityYoun-Bok Lee, Pranetha Baskaran, Jorge Gomez-Deza, et al.
Frontiers in Neuroscience|August 7, 2025
Accumulation of TDP-43 causes karyopherin-α4 pathology that characterises amyotrophic lateral sclerosisManpreet Singh Atwal, Jerneja Nimac, Urša Čerček, et al.
Molecular Cell|May 4, 2019
Cross-Regulation between TDP-43 and Paraspeckles Promotes Pluripotency-Differentiation TransitionMiha Modic, Markus Grosch, Gregor Rot, et al.
Biomedicines|June 28, 2023
Emerging Trends in the Field of Inflammation and Proteinopathy in ALS/FTD Spectrum DisorderFabiola De Marchi, Toni Franjkic, Paride Schito, et al.
Science (New York, N.Y.)|March 1, 2008
TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosisJemeen Sreedharan, Ian P Blair, Vineeta B Tripathi, et al.
Pageof 9

Showing results (61-70 of 82) with videos related to

Sort By:
Pageof 9
Nature Neuroscience|March 2, 2011
Characterizing the RNA targets and position-dependent splicing regulation by TDP-43James R Tollervey, Tomaž Curk, Boris Rogelj, et al.
Journal of Cell Science|February 13, 2019
Nuclear RNA foci from <i>C9ORF72</i> expansion mutation form paraspeckle-like bodiesAna Bajc Česnik, Simona Darovic, Sonja Prpar Mihevc, et al.
Plos One|March 22, 2014
Allele-specific knockdown of ALS-associated mutant TDP-43 in neural stem cells derived from induced pluripotent stem cellsAgnes L Nishimura, Carole Shum, Emma L Scotter, et al.
Human Molecular Genetics|October 4, 2017
C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicityYoun-Bok Lee, Pranetha Baskaran, Jorge Gomez-Deza, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|December 21, 2011
An MND/ALS phenotype associated with C9orf72 repeat expansion: abundant p62-positive, TDP-43-negative inclusions in cerebral cortex, hippocampus and cerebellum but without associated cognitive declineClaire Troakes, Satomi Maekawa, Lokesh Wijesekera, et al.
Human Molecular Genetics|September 5, 2020
C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicityYoun-Bok Lee, Pranetha Baskaran, Jorge Gomez-Deza, et al.
Frontiers in Neuroscience|August 7, 2025
Accumulation of TDP-43 causes karyopherin-α4 pathology that characterises amyotrophic lateral sclerosisManpreet Singh Atwal, Jerneja Nimac, Urša Čerček, et al.
Molecular Cell|May 4, 2019
Cross-Regulation between TDP-43 and Paraspeckles Promotes Pluripotency-Differentiation TransitionMiha Modic, Markus Grosch, Gregor Rot, et al.
Biomedicines|June 28, 2023
Emerging Trends in the Field of Inflammation and Proteinopathy in ALS/FTD Spectrum DisorderFabiola De Marchi, Toni Franjkic, Paride Schito, et al.
Science (New York, N.Y.)|March 1, 2008
TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosisJemeen Sreedharan, Ian P Blair, Vineeta B Tripathi, et al.
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