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Plos Genetics|October 10, 2007
Ancient exaptation of a CORE-SINE retroposon into a highly conserved mammalian neuronal enhancer of the proopiomelanocortin geneAndrea M Santangelo, Flávio S J de Souza, Lucía F Franchini, et al.Molecular Biology and Evolution|May 29, 2023
Accelerated Evolution Analysis Uncovers PKNOX2 as a Key Transcription Factor in the Mammalian CochleaAnabella P Trigila, Valeria C Castagna, Lara Berasain, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 26, 2008
Lmx1b-controlled isthmic organizer is essential for development of midbrain dopaminergic neuronsChao Guo, Hai-Yan Qiu, Ming Shi, et al.Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|November 13, 2013
Enhancer turnover and conserved regulatory function in vertebrate evolutionSabina Domené, Viviana F Bumaschny, Flávio S J de Souza, et al.Molecular Metabolism|October 1, 2016
Reprogramming the body weight set point by a reciprocal interaction of hypothalamic leptin sensitivity and Pomc gene expression reverts extreme obesityKavaljit H Chhabra, Jessica M Adams, Graham L Jones, et al.Proceedings of the National Academy of Sciences of the United States of America|April 1, 2015
Islet 1 specifies the identity of hypothalamic melanocortin neurons and is critical for normal food intake and adiposity in adulthoodSofia Nasif, Flavio S J de Souza, Laura E González, et al.Endocrinology|January 16, 2014
Selective disruption of dopamine D2 receptors in pituitary lactotropes increases body weight and adiposity in female miceMaria Ines Perez Millan, Guillermina Maria Luque, Maria Cecilia Ramirez, et al.Proceedings of the National Academy of Sciences of the United States of America|April 23, 2024
A mammalian tripartite enhancer cluster controls hypothalamic <i>Pomc</i> expression, food intake, and body weightDaniela Rojo, Clara E Hael, Agustina Soria, et al.Neuron|May 20, 2016
Enhanced GABA Transmission Drives Bradykinesia Following Loss of Dopamine D2 Receptor SignalingJulia C Lemos, Danielle M Friend, Alanna R Kaplan, et al.Endocrinology|March 18, 2003
Hypothalamic proopiomelanocortin neurons are glucose responsive and express K(ATP) channelsNurhadi Ibrahim, Martha A Bosch, James L Smart, et al.Pageof 11