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Nature|June 25, 2004
A membrane protein required for dislocation of misfolded proteins from the ERBrendan N Lilley, Hidde L PloeghImmunological Reviews|September 27, 2005
Viral modulation of antigen presentation: manipulation of cellular targets in the ER and beyondBrendan N Lilley, Hidde L PloeghProceedings of the National Academy of Sciences of the United States of America|September 28, 2005
Multiprotein complexes that link dislocation, ubiquitination, and extraction of misfolded proteins from the endoplasmic reticulum membraneBrendan N Lilley, Hidde L PloeghMolecular Biology of the Cell|September 16, 2003
Dislocation of a type I membrane protein requires interactions between membrane-spanning segments within the lipid bilayerBrendan N Lilley, Domenico Tortorella, Hidde L PloeghThe Journal of Cell Biology|October 18, 2006
SEL1L, the homologue of yeast Hrd3p, is involved in protein dislocation from the mammalian ERBritta Mueller, Brendan N Lilley, Hidde L PloeghNeuron|June 25, 2013
SAD kinases sculpt axonal arbors of sensory neurons through long- and short-term responses to neurotrophin signalsBrendan N Lilley, Y Albert Pan, Joshua R SanesJournal of Virology|August 17, 2006
Murine polyomavirus requires the endoplasmic reticulum protein Derlin-2 to initiate infectionBrendan N Lilley, Joanna M Gilbert, Hidde L Ploegh, et al.Proceedings of the National Academy of Sciences of the United States of America|November 13, 2013
Lkb1/Stk11 regulation of mTOR signaling controls the transition of chondrocyte fates and suppresses skeletal tumor formationLick Pui Lai, Brendan N Lilley, Joshua R Sanes, et al.Proceedings of the National Academy of Sciences of the United States of America|January 8, 2014
SAD kinases control the maturation of nerve terminals in the mammalian peripheral and central nervous systemsBrendan N Lilley, Arjun Krishnaswamy, Zhi Wang, et al.Nature|June 2, 2006
Signal peptide peptidase is required for dislocation from the endoplasmic reticulumJoana Loureiro, Brendan N Lilley, Eric Spooner, et al.Pageof 2