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Journal of Virology|February 15, 2005
Human cytomegalovirus protein US11 provokes an unfolded protein response that may facilitate the degradation of class I major histocompatibility complex productsBoaz Tirosh, Neal N Iwakoshi, Brendan N Lilley, et al.Neural Development|September 24, 2008
A chemical-genetic strategy reveals distinct temporal requirements for SAD-1 kinase in neuronal polarization and synapse formationJoanne S M Kim, Brendan N Lilley, Chao Zhang, et al.Cell|July 10, 2004
The small RNA chaperone Hfq and multiple small RNAs control quorum sensing in Vibrio harveyi and Vibrio choleraeDerrick H Lenz, Kenny C Mok, Brendan N Lilley, et al.Cellular Microbiology|June 15, 2005
Cellular internalization of cytolethal distending toxin: a new end to a known pathwayLina Guerra, Ken Teter, Brendan N Lilley, et al.Molecular and Cellular Biology|May 1, 2013
SAD-A potentiates glucose-stimulated insulin secretion as a mediator of glucagon-like peptide 1 response in pancreatic β cellsJia Nie, Brendan N Lilley, Y Albert Pan, et al.Molecular and Cellular Biology|January 12, 2011
Derlin-2-deficient mice reveal an essential role for protein dislocation in chondrocytesStephanie K Dougan, Chih-Chi Andrew Hu, Marie-Eve Paquet, et al.Cell|May 8, 2007
LKB1 and SAD kinases define a pathway required for the polarization of cortical neuronsAnthony P Barnes, Brendan N Lilley, Y Albert Pan, et al.The Journal of Comparative Neurology|August 5, 2018
Genetic access to neurons in the accessory optic system reveals a role for Sema6A in midbrain circuitry mediating motion perceptionBrendan N Lilley, Shai Sabbah, John L Hunyara, et al.Nature Neuroscience|August 4, 2014
LKB1 and AMPK regulate synaptic remodeling in old ageMelanie A Samuel, P Emanuela Voinescu, Brendan N Lilley, et al.Proceedings of the National Academy of Sciences of the United States of America|August 8, 2013
SAD-A kinase controls islet β-cell size and function as a mediator of mTORC1 signalingJia Nie, Xiaolei Liu, Brendan N Lilley, et al.Pageof 2