Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Brian Krug

Showing results (1-10 of 14) with videos related to

Pageof 2
Sort By:
The FEBS Journal|May 10, 2021
Oncohistones: a roadmap to stalled developmentShriya Deshmukh, Adam Ptack, Brian Krug, et al.
Trends in Cell Biology|June 7, 2021
Polycomb repressive complex 2 in the driver's seat of childhood and young adult brain tumoursBrian Krug, Ashot S Harutyunyan, Shriya Deshmukh, et al.
Cell Reports|November 18, 2020
H3K27M in Gliomas Causes a One-Step Decrease in H3K27 Methylation and Reduced Spreading within the Constraints of H3K36 MethylationAshot S Harutyunyan, Haifen Chen, Tianyuan Lu, et al.
Molecular Cell|November 5, 2021
Histone H3.3 K27M and K36M mutations de-repress transposable elements through perturbation of antagonistic chromatin marksAmel Chaouch, Johannes Berlandi, Carol C L Chen, et al.
Genome Biology|December 9, 2023
Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodiesHsiao P J Voon, Linda Hii, Andrew Garvie, et al.
Nature Communications|July 22, 2025
Aberrant EZHIP expression drives tumorigenesis in osteosarcomaWajih Jawhar, Geoffroy Danieau, Alva Annett, et al.
Cancer Cell|May 16, 2019
Pervasive H3K27 Acetylation Leads to ERV Expression and a Therapeutic Vulnerability in H3K27M GliomasBrian Krug, Nicolas De Jay, Ashot S Harutyunyan, et al.
Biorxiv : the Preprint Server for Biology|December 20, 2023
H3K27me3 spreading organizes canonical PRC1 chromatin architecture to regulate developmental programsBrian Krug, Bo Hu, Haifen Chen, et al.
Nature Communications|March 21, 2019
H3K27M induces defective chromatin spread of PRC2-mediated repressive H3K27me2/me3 and is essential for glioma tumorigenesisAshot S Harutyunyan, Brian Krug, Haifen Chen, et al.
Nature Genetics|December 5, 2022
K27M in canonical and noncanonical H3 variants occurs in distinct oligodendroglial cell lineages in brain midline gliomasSelin Jessa, Abdulshakour Mohammadnia, Ashot S Harutyunyan, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
The FEBS Journal|May 10, 2021
Oncohistones: a roadmap to stalled developmentShriya Deshmukh, Adam Ptack, Brian Krug, et al.
Trends in Cell Biology|June 7, 2021
Polycomb repressive complex 2 in the driver's seat of childhood and young adult brain tumoursBrian Krug, Ashot S Harutyunyan, Shriya Deshmukh, et al.
Cell Reports|November 18, 2020
H3K27M in Gliomas Causes a One-Step Decrease in H3K27 Methylation and Reduced Spreading within the Constraints of H3K36 MethylationAshot S Harutyunyan, Haifen Chen, Tianyuan Lu, et al.
Molecular Cell|November 5, 2021
Histone H3.3 K27M and K36M mutations de-repress transposable elements through perturbation of antagonistic chromatin marksAmel Chaouch, Johannes Berlandi, Carol C L Chen, et al.
Genome Biology|December 9, 2023
Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodiesHsiao P J Voon, Linda Hii, Andrew Garvie, et al.
Nature Communications|July 22, 2025
Aberrant EZHIP expression drives tumorigenesis in osteosarcomaWajih Jawhar, Geoffroy Danieau, Alva Annett, et al.
Cancer Cell|May 16, 2019
Pervasive H3K27 Acetylation Leads to ERV Expression and a Therapeutic Vulnerability in H3K27M GliomasBrian Krug, Nicolas De Jay, Ashot S Harutyunyan, et al.
Biorxiv : the Preprint Server for Biology|December 20, 2023
H3K27me3 spreading organizes canonical PRC1 chromatin architecture to regulate developmental programsBrian Krug, Bo Hu, Haifen Chen, et al.
Nature Communications|March 21, 2019
H3K27M induces defective chromatin spread of PRC2-mediated repressive H3K27me2/me3 and is essential for glioma tumorigenesisAshot S Harutyunyan, Brian Krug, Haifen Chen, et al.
Nature Genetics|December 5, 2022
K27M in canonical and noncanonical H3 variants occurs in distinct oligodendroglial cell lineages in brain midline gliomasSelin Jessa, Abdulshakour Mohammadnia, Ashot S Harutyunyan, et al.
Pageof 2