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Scientific Reports|January 29, 2021
Inhibition of mitochondrial function by metformin increases glucose uptake, glycolysis and GDF-15 release from intestinal cellsMing Yang, Tamana Darwish, Pierre Larraufie, et al.
Cell Metabolism|July 12, 2023
Fetal manipulation of maternal metabolism is a critical function of the imprinted Igf2 geneJorge Lopez-Tello, Hannah E J Yong, Ionel Sandovici, et al.
Molecular Metabolism|May 3, 2017
Heterogeneity of hypothalamic pro-opiomelanocortin-expressing neurons revealed by single-cell RNA sequencingBrian Y H Lam, Irene Cimino, Joseph Polex-Wolf, et al.
Plos Genetics|October 15, 2020
Mesenchyme-derived IGF2 is a major paracrine regulator of pancreatic growth and functionConstanze M Hammerle, Ionel Sandovici, Gemma V Brierley, et al.
Circulation Research|October 16, 2019
Impaired Autophagy in CD11b+ Dendritic Cells Expands CD4+ Regulatory T Cells and Limits Atherosclerosis in MiceMarc Clement, Juliette Raffort, Fabien Lareyre, et al.
Redox Biology|March 21, 2021
Sirt3 in POMC neurons controls energy balance in a sex- and diet-dependent mannerMar Quiñones, René Hernández-Bautista, Daniel Beiroa, et al.
Molecular Metabolism|January 2, 2023
Obesity medication lorcaserin activates brainstem GLP-1 neurons to reduce food intake and augments GLP-1 receptor agonist induced appetite suppressionStefan Wagner, Daniel I Brierley, Alasdair Leeson-Payne, et al.
Nature Metabolism|September 17, 2021
SREBP1-induced fatty acid synthesis depletes macrophages antioxidant defences to promote their alternative activationGuillaume Bidault, Samuel Virtue, Kasparas Petkevicius, et al.
Nature Communications|May 29, 2023
Dax1 modulates ERα-dependent hypothalamic estrogen sensing in female miceJose M Ramos-Pittol, Isabel Fernandes-Freitas, Alexandra Milona, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 17, 2017
Obesity-associated gene TMEM18 has a role in the central control of appetite and body weight regulationRachel Larder, M F Michelle Sim, Pawan Gulati, et al.
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