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Frontiers in Molecular Neuroscience|October 3, 2022
Breakdown of the central synapses in C9orf72-linked ALS/FTDLayla T Ghaffari, Davide Trotti, Aaron R Haeusler, et al.
Brain Research|August 24, 2019
White matter loss and oligodendrocyte dysfunction in HIV: A consequence of the infection, the antiretroviral therapy or both?Brigid K Jensen, Lindsay M Roth, Judith B Grinspan, et al.
Cell Reports|October 13, 2016
Cell-to-Cell Transmission of Dipeptide Repeat Proteins Linked to C9orf72-ALS/FTDThomas Westergard, Brigid K Jensen, Xinmei Wen, et al.
Journal of Neuroimmune Pharmacology : the Official Journal of the Society on Neuroimmune Pharmacology|November 29, 2019
Protease Inhibitors, Saquinavir and Darunavir, Inhibit Oligodendrocyte Maturation: Implications for Lysosomal StressLindsay Festa, Lindsay M Roth, Brigid K Jensen, et al.
Biorxiv : the Preprint Server for Biology|February 23, 2026
Intrathecal (G 4 C 2 ) 149 delivery in C9orf72-deficient mice yields mild motor dysfunction and ALS/FTD pathological hallmarksKatelyn A Russell, Amelia A Shahrabi, Suleyman C Akerman, et al.
Acta Neuropathologica Communications|June 19, 2026
Intrathecal (G4C2)149 delivery in C9orf72-deficient mice yields mild motor dysfunction and ALS/FTD pathological hallmarksKatelyn A Russell, Amelia A Shahrabi, Suleyman C Akerman, et al.
Scientific Reports|April 5, 2022
A mouse model with widespread expression of the C9orf72-linked glycine-arginine dipeptide displays non-lethal ALS/FTD-like phenotypesBrandie Morris Verdone, Maria Elena Cicardi, Xinmei Wen, et al.
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