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Journal of Medical Genetics|May 8, 2007
Phenotypic diversity of Menkes disease in mottled mice is associated with defects in localisation and trafficking of the ATP7A proteinByung-Eun Kim, Michael J Petris
Frontiers in Cell and Developmental Biology|April 18, 2022
Oral Elesclomol Treatment Alleviates Copper Deficiency in Animal ModelsSai Yuan, Tamara Korolnek, Byung-Eun Kim
Nature Chemical Biology|February 19, 2008
Mechanisms for copper acquisition, distribution and regulationByung-Eun Kim, Tracy Nevitt, Dennis J Thiele
Cell Metabolism|September 5, 2006
Ctr1 drives intestinal copper absorption and is essential for growth, iron metabolism, and neonatal cardiac functionYasuhiro Nose, Byung-Eun Kim, Dennis J Thiele
The Journal of Biological Chemistry|August 24, 2004
The mammalian Zip5 protein is a zinc transporter that localizes to the basolateral surface of polarized cellsFudi Wang, Byung-Eun Kim, Michael J Petris, et al.
Scientific Reports|September 22, 2017
Organ-specific regulation of ATP7A abundance is coordinated with systemic copper homeostasisHaarin Chun, Tracy Catterton, Heejeong Kim, et al.
The Journal of Biological Chemistry|September 11, 2002
A conditional mutation affecting localization of the Menkes disease copper ATPase. Suppression by copper supplementationByung-Eun Kim, Kathryn Smith, Carisa K Meagher, et al.
The Journal of Biological Chemistry|May 23, 2018
CHCA-1 is a copper-regulated CTR1 homolog required for normal development, copper accumulation, and copper-sensing behavior in <i>Caenorhabditis elegans</i>Sai Yuan, Anuj Kumar Sharma, Alexandria Richart, et al.
The Journal of Biological Chemistry|January 5, 2007
A histidine-rich cluster mediates the ubiquitination and degradation of the human zinc transporter, hZIP4, and protects against zinc cytotoxicityXiaoqing Mao, Byung-Eun Kim, Fudi Wang, et al.
The Journal of Biological Chemistry|November 25, 2016
The Intestinal Copper Exporter CUA-1 Is Required for Systemic Copper Homeostasis in Caenorhabditis elegansHaarin Chun, Anuj Kumar Sharma, Jaekwon Lee, et al.
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