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C A Finlay

Showing results (1-10 of 13) with videos related to

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Molecular and Cellular Biology|January 1, 1993
The mdm-2 oncogene can overcome wild-type p53 suppression of transformed cell growthC A Finlay
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|August 1, 1992
p53 loss of function: implications for the processes of immortalization and tumorigenesisC A Finlay
Experimental Cell Research|January 1, 1987
Autocrine stimulation of WI38 cell proliferation in the presence of glucocorticoids. Characteristics of the stimulatory factor(s) involved in this responseC A Finlay, V J Cristofalo
Oncogene|February 1, 1993
A functionally inactive p53 Li-Fraumeni syndrome mutantM Hao, C A Finlay, G Lozano
Nature|June 6, 1991
The p53 tumour suppressor geneA J Levine, J Momand, C A Finlay
Cell|June 30, 1989
The p53 proto-oncogene can act as a suppressor of transformationC A Finlay, P W Hinds, A J Levine
Molecular and Cellular Biology|August 1, 1987
Immunological evidence for the association of p53 with a heat shock protein, hsc70, in p53-plus-ras-transformed cell linesP W Hinds, C A Finlay, A B Frey, et al.
Experimental Cell Research|February 1, 1985
Enhanced proliferation of WI38 cells in the presence of glucocorticoid-conditioned mediumC A Finlay, B A Rosner, J E Miller, et al.
Molecular and Cellular Biology|February 1, 1988
Activating mutations for transformation by p53 produce a gene product that forms an hsc70-p53 complex with an altered half-lifeC A Finlay, P W Hinds, T H Tan, et al.
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|April 1, 1999
Overproduction of MDM2 in vivo disrupts S phase independent of E2F1V Reinke, D M Bortner, L L Amelse, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
Molecular and Cellular Biology|January 1, 1993
The mdm-2 oncogene can overcome wild-type p53 suppression of transformed cell growthC A Finlay
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|August 1, 1992
p53 loss of function: implications for the processes of immortalization and tumorigenesisC A Finlay
Experimental Cell Research|January 1, 1987
Autocrine stimulation of WI38 cell proliferation in the presence of glucocorticoids. Characteristics of the stimulatory factor(s) involved in this responseC A Finlay, V J Cristofalo
Oncogene|February 1, 1993
A functionally inactive p53 Li-Fraumeni syndrome mutantM Hao, C A Finlay, G Lozano
Nature|June 6, 1991
The p53 tumour suppressor geneA J Levine, J Momand, C A Finlay
Cell|June 30, 1989
The p53 proto-oncogene can act as a suppressor of transformationC A Finlay, P W Hinds, A J Levine
Molecular and Cellular Biology|August 1, 1987
Immunological evidence for the association of p53 with a heat shock protein, hsc70, in p53-plus-ras-transformed cell linesP W Hinds, C A Finlay, A B Frey, et al.
Experimental Cell Research|February 1, 1985
Enhanced proliferation of WI38 cells in the presence of glucocorticoid-conditioned mediumC A Finlay, B A Rosner, J E Miller, et al.
Molecular and Cellular Biology|February 1, 1988
Activating mutations for transformation by p53 produce a gene product that forms an hsc70-p53 complex with an altered half-lifeC A Finlay, P W Hinds, T H Tan, et al.
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|April 1, 1999
Overproduction of MDM2 in vivo disrupts S phase independent of E2F1V Reinke, D M Bortner, L L Amelse, et al.
Pageof 2