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American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|December 19, 2003
Novel CAG/CTG repeat expansion mutations do not contribute to the genetic risk for most cases of bipolar disorder or schizophreniaT Tsutsumi, S E Holmes, M G McInnis, et al.
Neurology|February 1, 1997
Longitudinal change in basal ganglia volume in patients with Huntington's diseaseE H Aylward, Q Li, O C Stine, et al.
Nature|November 23, 1995
A huntingtin-associated protein enriched in brain with implications for pathologyX J Li, S H Li, A H Sharp, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 1, 1984
Tonic vasomotor control by the rostral ventrolateral medulla: effect of electrical or chemical stimulation of the area containing C1 adrenaline neurons on arterial pressure, heart rate, and plasma catecholamines and vasopressinC A Ross, D A Ruggiero, D H Park, et al.
The Journal of Cell Biology|September 7, 2000
Atrophin-1, the dentato-rubral and pallido-luysian atrophy gene product, interacts with ETO/MTG8 in the nuclear matrix and represses transcriptionJ D Wood, F C Nucifora, K Duan, et al.
Nature Genetics|July 1, 1995
Microsatellite evolution--evidence for directionality and variation in rate between speciesD C Rubinsztein, W Amos, J Leggo, et al.
Brain Research. Molecular Brain Research|March 1, 1996
DRPLA gene (atrophin-1) sequence and mRNA expression in human brainR L Margolis, S H Li, W S Young, et al.
Journal of Medical Genetics|February 9, 1999
Unstable expansion of the CAG trinucleotide repeat in MAB21L1: report of a second pedigree and effect on protein expressionR L Margolis, O C Stine, C M Ward, et al.
Molecular and Cellular Neurosciences|July 2, 1998
Atrophin-1, the DRPLA gene product, interacts with two families of WW domain-containing proteinsJ D Wood, J Yuan, R L Margolis, et al.
Human Molecular Genetics|April 20, 2001
Inducible expression of mutant alpha-synuclein decreases proteasome activity and increases sensitivity to mitochondria-dependent apoptosisY Tanaka, S Engelender, S Igarashi, et al.
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