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Human Genetics|February 1, 1996
Psychosis and genes with trinucleotide repeat polymorphismT Sasaki, E Billett, A Petronis, et al.Neurobiology of Disease|July 13, 2001
Creatine increase survival and delays motor symptoms in a transgenic animal model of Huntington's diseaseO A Andreassen, A Dedeoglu, R J Ferrante, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 11, 2000
Wild-type huntingtin protects from apoptosis upstream of caspase-3D Rigamonti, J H Bauer, C De-Fraja, et al.Human Molecular Genetics|May 23, 1998
Truncated N-terminal fragments of huntingtin with expanded glutamine repeats form nuclear and cytoplasmic aggregates in cell cultureJ K Cooper, G Schilling, M F Peters, et al.Nature Genetics|May 13, 1999
Synphilin-1 associates with alpha-synuclein and promotes the formation of cytosolic inclusionsS Engelender, Z Kaminsky, X Guo, et al.The Journal of Biological Chemistry|March 20, 1999
Cleavage of atrophin-1 at caspase site aspartic acid 109 modulates cytotoxicityL M Ellerby, R L Andrusiak, C L Wellington, et al.Molecular Psychiatry|August 14, 1998
Large CAG/CTG repeats are associated with childhood-onset schizophreniaC E Burgess, K Lindblad, E Sidransky, et al.Journal of Neurology, Neurosurgery, and Psychiatry|December 22, 2007
Detection of Huntington's disease decades before diagnosis: the Predict-HD studyJ S Paulsen, D R Langbehn, J C Stout, et al.Movement Disorders : Official Journal of the Movement Disorder Society|July 1, 1997
Dentatorubral and pallidoluysian atrophy (DRPLA). Clinical and neuropathological findings in genetically confirmed North American and European pedigreesM W Becher, D C Rubinsztein, J Leggo, et al.Human Molecular Genetics|February 9, 1999
Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtinG Schilling, M W Becher, A H Sharp, et al.Pageof 32