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The Netherlands Journal of Medicine|October 6, 2004
Ocular syphilis acquired through oral sex in two HIV-infected patientsM B B McCall, J J C van Lith-Verhoeven, R van Crevel, et al.The British Journal of Ophthalmology|May 29, 1998
Increased presence of Epstein-Barr virus DNA in ocular fluid samples from HIV negative immunocompromised patients with uveitisJ V Ongkosuwito, A Van der Lelij, M Bruinenberg, et al.Clinical Genetics|June 12, 2018
Genotype-phenotype correlations of low-frequency variants in the complement system in renal disease and age-related macular degenerationM J Geerlings, E B Volokhina, E K de Jong, et al.Ophthalmic Genetics|March 23, 2002
Clinical characterization, linkage analysis, and PRPC8 mutation analysis of a family with autosomal dominant retinitis pigmentosa type 13 (RP13)J J C van Lith-Verhoeven, S D van der Velde-Visser, M M Sohocki, et al.Genomics|June 22, 1999
Isolation and mapping of novel candidate genes for retinal disorders using suppression subtractive hybridizationA I den Hollander, M A van Driel, Y J de Kok, et al.BMC Ophthalmology|July 8, 2015
Comparing the effectiveness and costs of Bevacizumab to Ranibizumab in patients with Diabetic Macular Edema: a randomized clinical trial (the BRDME study)A M E Schauwvlieghe, G Dijkman, J M Hooymans, et al.Human Molecular Genetics|April 18, 1998
Autosomal recessive retinitis pigmentosa and cone-rod dystrophy caused by splice site mutations in the Stargardt's disease gene ABCRF P Cremers, D J van de Pol, M van Driel, et al.American Journal of Human Genetics|March 26, 1999
The 2588G-->C mutation in the ABCR gene is a mild frequent founder mutation in the Western European population and allows the classification of ABCR mutations in patients with Stargardt diseaseA Maugeri, M A van Driel, D J van de Pol, et al.Human Molecular Genetics|July 27, 2001
Mutations in the pre-mRNA splicing factor gene PRPC8 in autosomal dominant retinitis pigmentosa (RP13)A B McKie, J C McHale, T J Keen, et al.Clinical Genetics|April 21, 2018
Further audiovestibular characterization of DFNB77, caused by deleterious variants in LOXHD1, and investigation into the involvement of Fuchs corneal dystrophyM Wesdorp, V Schreur, A J Beynon, et al.Pageof 5