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Blood|January 12, 2012
p38 MAPK inhibition suppresses the TLR-hypersensitive phenotype in FANCC- and FANCA-deficient mononuclear phagocytesPraveen Anur, Jane Yates, Michael R Garbati, et al.Trends in Microbiology|September 9, 2023
What is microbial dormancy?Mark D McDonald, Carlos Owusu-Ansah, Jared B Ellenbogen, et al.Blood|September 10, 2010
Fancd2-/- mice have hematopoietic defects that can be partially corrected by resveratrolQing-Shuo Zhang, Laura Marquez-Loza, Laura Eaton, et al.Blood|June 2, 2012
FANCL ubiquitinates β-catenin and enhances its nuclear functionKim-Hien T Dao, Michael D Rotelli, Curtis L Petersen, et al.Blood|July 1, 1996
Germ cell defects and hematopoietic hypersensitivity to gamma-interferon in mice with a targeted disruption of the Fanconi anemia C geneM A Whitney, G Royle, M J Low, et al.Experimental Hematology|January 16, 1998
Expression of the Fanconi anemia group C gene in hematopoietic cells is not influenced by oxidative stress, cross-linking agents, radiation, heat, or mitotic inhibitory factorsP A Tower, T A Christianson, S T Peters, et al.Blood|October 24, 2009
TLR8-dependent TNF-(alpha) overexpression in Fanconi anemia group C cellsScott M Vanderwerf, Johanna Svahn, Susan Olson, et al.Nature Structural & Molecular Biology|April 26, 2011
Conformational changes in IgE contribute to its uniquely slow dissociation rate from receptor FcɛRIMary D Holdom, Anna M Davies, Joanne E Nettleship, et al.Scientific Reports|January 20, 2025
A climatically significant abiotic mechanism driving carbon loss and nitrogen limitation in peat bogsAlexandra B Cory, Rachel M Wilson, M Elizabeth Holmes, et al.Blood|August 1, 1997
Inactivation of the Fanconi anemia group C gene augments interferon-gamma-induced apoptotic responses in hematopoietic cellsR K Rathbun, G R Faulkner, M H Ostroski, et al.Pageof 14