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Molecular Endocrinology (Baltimore, Md.)|November 1, 1994
A Pit-1 phosphorylation mutant can mediate both basal and induced prolactin and growth hormone promoter activityD J Fischberg, X H Chen, C Bancroft
Talanta|January 1, 1985
Arsenic speciation in soil-pore waters from mineralized and unmineralized areas of south-west EnglandS J Haswell, P O'neill, K C Bancroft
Plos One|April 13, 2012
Assessing public engagement with science in a university primate research centre in a national zooMark T Bowler, Hannah M Buchanan-Smith, Andrew Whiten
The Journal of Biological Chemistry|June 25, 1981
Calcium specifically stimulates prolactin synthesis and messenger RNA sequences in GH3 cellsB A White, L R Bauerle, F C Bancroft
Endocrinology|April 1, 1996
Constitutively active G(S) alpha-subunits stimulate Pit-1 promoter activity via a protein kinase A-mediated pathway acting through deoxyribonucleic acid binding sites both for Pit-1 and for adenosine 3',5'-monophosphate response element-binding proteinC Gaiddon, J Tian, J P Loeffler, et al.
The Journal of Biological Chemistry|June 10, 1977
Glucocorticoid induction of growth hormone synthesis in a strain of rat pituitary cellsL Y Yu, R J Tushinski, F C Bancroft
The Journal of Biological Chemistry|May 25, 1979
Interferon priming. Effects on interferon messenger RNAS L Abreu, F C Bancroft, W E Stewart
Proceedings of the National Academy of Sciences of the United States of America|January 1, 1976
Pregrowth hormone: product of the translation in vitro of messenger RNA coding for growth hormoneP M Sussman, R J Tushinski, F C Bancroft
Endocrinology|October 1, 1995
Transcriptional effects in GH3 cells of Gs alpha mutants associated with human pituitary tumors: stimulation of adenosine 3',5'-monophosphate response element-binding protein-mediated transcription and of prolactin and growth hormone promoter activity via protein kinase AC Gaiddon, L Mercken, C Bancroft, et al.
Endocrinology|April 1, 1996
Pituitary adenylate cyclase-activating polypeptide regulates prolactin promoter activity via a protein kinase A-mediated pathway that is independent of the transcriptional pathway employed by thyrotropin-releasing hormoneD T Coleman, X Chen, M Sassaroli, et al.
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