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The Journal of Experimental Medicine|January 20, 1999
Mitochondrial release of caspase-2 and -9 during the apoptotic processS A Susin, H K Lorenzo, N Zamzami, et al.Oncogene|January 25, 2000
Oxidation of a critical thiol residue of the adenine nucleotide translocator enforces Bcl-2-independent permeability transition pore opening and apoptosisP Costantini, A S Belzacq, H L Vieira, et al.The Journal of Experimental Medicine|May 23, 1998
The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteinsI Marzo, C Brenner, N Zamzami, et al.Cellular and Molecular Life Sciences : CMLS|February 24, 2001
Pathophysiology of mitochondrial cell death controlH L Vieira, G KroemerNature Cell Biology|November 21, 2001
Organelle-specific initiation of cell death pathwaysK F Ferri, G KroemerZeitschrift Fur Die Gesamte Innere Medizin Und Ihre Grenzgebiete|January 15, 1988
[Concomitant psychotherapy treatment of bronchial asthma]H F Böttcher, G KroemerBioessays : News and Reviews in Molecular, Cellular and Developmental Biology|February 13, 2001
Mitochondria--the suicide organellesK F Ferri, G KroemerOncogene|February 3, 2000
Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocatorC Brenner, H Cadiou, H L Vieira, et al.Pageof 48