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C D'Sa

Showing results (11-20 of 20) with videos related to

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Journal of Neuroscience Methods|November 1, 1996
Tryptophan hydroxylase: purification by affinity chromatography on calmodulin-sepharoseC D'Sa, R Arthur, I Jennings, et al.
Brain Research. Molecular Brain Research|September 5, 1996
Tetrahydrobiopterin biosynthesis in C6 glioma cells: induction of GTP cyclohydrolase I gene expression by lipopolysaccharide and cytokine treatmentC D'Sa, K Hirayama, A West, et al.
Brain Research. Developmental Brain Research|June 20, 2001
Bid regulation of neuronal apoptosisJ R Leonard, C D'Sa, B R Cahn, et al.
Cancer Research|June 1, 1996
Deletion of a nonconserved region of Bcl-2 confers a novel gain of function: suppression of apoptosis with concomitant cell proliferationE J Uhlmann, C D'Sa-Eipper, T Subramanian, et al.
Journal of Neuroscience Research|July 15, 2009
Inactivation of fibroblast growth factor receptor signaling in myelinating glial cells results in significant loss of adult spiral ganglion neurons accompanied by age-related hearing impairmentS J Wang, M Furusho, C D'Sa, et al.
Cell|October 21, 1994
Adenovirus E1B 19 kDa and Bcl-2 proteins interact with a common set of cellular proteinsJ M Boyd, S Malstrom, T Subramanian, et al.
Brain Research|October 9, 1995
Neurokinin-3 receptors modulate dopamine cell function and alter the effects of 6-hydroxydopamineM J Bannon, L A Brownschidle, Y Tian, et al.
Development (Cambridge, England)|November 28, 2000
DNA damage-induced neural precursor cell apoptosis requires p53 and caspase 9 but neither Bax nor caspase 3C D'Sa-Eipper, J R Leonard, G Putcha, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 11, 2001
Bcl-X(L)-caspase-9 interactions in the developing nervous system: evidence for multiple death pathwaysA U Zaidi, C D'Sa-Eipper, J Brenner, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 5, 2000
Epistatic and independent functions of caspase-3 and Bcl-X(L) in developmental programmed cell deathK A Roth, C Kuan, T F Haydar, et al.
Pageof 2

Showing results (11-20 of 20) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 20 results.
Journal of Neuroscience Methods|November 1, 1996
Tryptophan hydroxylase: purification by affinity chromatography on calmodulin-sepharoseC D'Sa, R Arthur, I Jennings, et al.
Brain Research. Molecular Brain Research|September 5, 1996
Tetrahydrobiopterin biosynthesis in C6 glioma cells: induction of GTP cyclohydrolase I gene expression by lipopolysaccharide and cytokine treatmentC D'Sa, K Hirayama, A West, et al.
Brain Research. Developmental Brain Research|June 20, 2001
Bid regulation of neuronal apoptosisJ R Leonard, C D'Sa, B R Cahn, et al.
Cancer Research|June 1, 1996
Deletion of a nonconserved region of Bcl-2 confers a novel gain of function: suppression of apoptosis with concomitant cell proliferationE J Uhlmann, C D'Sa-Eipper, T Subramanian, et al.
Journal of Neuroscience Research|July 15, 2009
Inactivation of fibroblast growth factor receptor signaling in myelinating glial cells results in significant loss of adult spiral ganglion neurons accompanied by age-related hearing impairmentS J Wang, M Furusho, C D'Sa, et al.
Cell|October 21, 1994
Adenovirus E1B 19 kDa and Bcl-2 proteins interact with a common set of cellular proteinsJ M Boyd, S Malstrom, T Subramanian, et al.
Brain Research|October 9, 1995
Neurokinin-3 receptors modulate dopamine cell function and alter the effects of 6-hydroxydopamineM J Bannon, L A Brownschidle, Y Tian, et al.
Development (Cambridge, England)|November 28, 2000
DNA damage-induced neural precursor cell apoptosis requires p53 and caspase 9 but neither Bax nor caspase 3C D'Sa-Eipper, J R Leonard, G Putcha, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 11, 2001
Bcl-X(L)-caspase-9 interactions in the developing nervous system: evidence for multiple death pathwaysA U Zaidi, C D'Sa-Eipper, J Brenner, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 5, 2000
Epistatic and independent functions of caspase-3 and Bcl-X(L) in developmental programmed cell deathK A Roth, C Kuan, T F Haydar, et al.
Pageof 2