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Journal of Neuroimmunology|February 1, 1988
Low-dose cyclosporin A induces relapsing remitting experimental allergic encephalomyelitis in the Lewis ratC H Polman, I Matthaei, C J de Groot, et al.Neurological Sciences : Official Journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology|October 18, 2001
Stress, glucocorticoids and the susceptibility to develop autoimmune disorders of the central nervous systemC Morale, J Brouwer, N Testa, et al.The American Journal of Pathology|October 1, 1993
Localization of beta 1 integrins and their extracellular ligands in human lymphoid tissuesT K van den Berg, M van der Ende, E A Döpp, et al.Journal of Neuropathology and Experimental Neurology|January 1, 1997
Immunocytochemical characterization of the expression of inducible and constitutive isoforms of nitric oxide synthase in demyelinating multiple sclerosis lesionsC J De Groot, S R Ruuls, J W Theeuwes, et al.Journal of Neuroimmunology|June 1, 1989
Observer agreement in the assessment of clinical signs in experimental allergic encephalomyelitisI Matthaei, C H Polman, C J de Groot, et al.The International Journal of Neuroscience|July 1, 1991
Interferon-gamma induced IA antigen expression on cultured neuroglial cells and brain macrophages from rat spinal cord and cerebrumC J De Groot, T Sminia, C D Dijkstra, et al.Clinical and Experimental Immunology|September 1, 1996
Susceptibility to clinically manifest cyclosporine A (CsA)-induced autoimmune disease is associated with interferon-gamma (IFN-gamma)-producing CD45RC+RT6- T helper cellsL J Beijleveld, H Groen, C P Broeren, et al.Clinical and Experimental Immunology|August 1, 1995
Anti-CD11b/CD18 antibodies reduce inflammation in acute colitis in ratsM J Palmen, C D Dijkstra, M B van der Ende, et al.Glia|September 11, 2010
The release of cytokines by macrophages is not affected by myelin ingestionJ E Glim, E J F Vereyken, D A M Heijnen, et al.Brain Research. Brain Research Reviews|December 18, 2001
Stress, the immune system and vulnerability to degenerative disorders of the central nervous system in transgenic mice expressing glucocorticoid receptor antisense RNAB Marchetti, M C Morale, N Testa, et al.Pageof 17