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The Journal of Biological Chemistry|February 15, 1986
Glucocorticoids increase cholecystokinin receptors and amylase secretion in pancreatic acinar AR42J cellsC D LogsdonLife Sciences|March 9, 1999
The influence of the cellular context on receptor function: a necessary consideration for physiologic interpretations of receptor expression studiesC D LogsdonThe American Journal of Physiology|October 1, 1986
Stimulation of pancreatic acinar cell growth by CCK, epidermal growth factor, and insulin in vitroC D LogsdonGut|January 1, 1987
Effects of calcium mediated secretagogues on the growth of pancreatic acinar cells in vitroC D LogsdonCurrent Opinion in Gastroenterology|October 13, 2006
Signal transduction in pancreatic acinar cell physiology and pathophysiologyC D LogsdonGastroenterology|October 1, 1991
Glucocorticoids stimulate ornithine decarboxylase gene expression in pancreatic AR42J cellsS Rosewicz, C D LogsdonThe American Journal of Physiology|December 1, 1993
Both low- and high-affinity CCK receptor states mediate trophic effects on rat pancreatic acinar cellsH Hoshi, C D LogsdonThe American Journal of Physiology|September 1, 1992
CCK, bombesin, and carbachol stimulate c-fos, c-jun, and c-myc oncogene expression in rat pancreatic aciniL Lu, C D LogsdonThe American Journal of Physiology|July 17, 1999
Cholecystokinin induction of mob-1 chemokine expression in pancreatic acinar cells requires NF-kappaB activationB Han, C D LogsdonBiochemical and Biophysical Research Communications|November 15, 1993
Direct trophic effects of fibroblast growth factors on rat pancreatic acinar cells in vitroH Hoshi, C D LogsdonPageof 8