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Molecular Pharmacology|December 1, 1993
Human m3 muscarinic acetylcholine receptor carboxyl-terminal threonine resides are required for agonist-induced receptor down-regulationJ Yang, C D Logsdon, T E Johansen, et al.
The American Journal of Physiology|August 1, 1984
Regulation of pancreatic acinar cell insulin receptors by insulinJ Mössner, C D Logsdon, I D Goldfine, et al.
The Journal of Cell Biology|April 1, 1985
Glucocorticoids increase amylase mRNA levels, secretory organelles, and secretion in pancreatic acinar AR42J cellsC D Logsdon, J Moessner, J A Williams, et al.
Digestion|January 1, 1997
Stimulation of both CCK-A and CCK-B receptors activates MAP kinases in AR42J and receptor-transfected CHO cellsA Dabrowski, K M Detjen, C D Logsdon, et al.
Gut|January 1, 1987
Do insulin and the insulin like growth factors (IGFs) stimulate growth of the exocrine pancreas?J Mössner, C D Logsdon, I D Goldfine, et al.
Molecular Pharmacology|September 1, 1995
Mutation of carboxyl-terminal threonine residues in human m3 muscarinic acetylcholine receptor modulates the extent of sequestration and desensitizationJ Yang, J A Williams, D I Yule, et al.
Diabetes|September 1, 1985
Insulin, via its own receptor, regulates growth and amylase synthesis in pancreatic acinar AR42J cellsJ Mössner, C D Logsdon, J A Williams, et al.
The American Journal of Physiology|December 1, 1988
Effects of cholecystokinin on pancreatic ornithine decarboxylase gene expressionS Rosewicz, L D Lewis, R A Liddle, et al.
The American Journal of Physiology|September 1, 1989
Identification of a 185-kDa band 3-related polypeptide in oxyntic cellsH A Thomas, T E Machen, A Smolka, et al.
Pflugers Archiv : European Journal of Physiology|November 1, 1992
The outwardly rectifying Cl- channel is not involved in cAMP-mediated Cl- secretion in HT-29 cells: evidence for a very-low-conductance Cl- channelH Fischer, K M Kreusel, B Illek, et al.
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