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Gastroenterology|December 12, 1997
Chemokine gene expression in rat pancreatic acinar cells is an early event associated with acute pancreatitisT Grady, P Liang, S A Ernst, et al.
Biochemical and Biophysical Research Communications|October 3, 1996
Stress-activated protein kinase activation is the earliest direct correlate to the induction of secretagogue-induced pancreatitis in ratsT Grady, A Dabrowski, J A Williams, et al.
The American Journal of Physiology|February 10, 1999
Adenovirus-mediated gene transfer of RasN17 inhibits specific CCK actions on pancreatic acinar cellsB Nicke, M J Tseng, M Fenrich, et al.
Molecular Pharmacology|December 1, 1993
Human m3 muscarinic acetylcholine receptor carboxyl-terminal threonine resides are required for agonist-induced receptor down-regulationJ Yang, C D Logsdon, T E Johansen, et al.
The American Journal of Physiology|August 1, 1984
Regulation of pancreatic acinar cell insulin receptors by insulinJ Mössner, C D Logsdon, I D Goldfine, et al.
The Journal of Cell Biology|April 1, 1985
Glucocorticoids increase amylase mRNA levels, secretory organelles, and secretion in pancreatic acinar AR42J cellsC D Logsdon, J Moessner, J A Williams, et al.
Digestion|January 1, 1997
Stimulation of both CCK-A and CCK-B receptors activates MAP kinases in AR42J and receptor-transfected CHO cellsA Dabrowski, K M Detjen, C D Logsdon, et al.
Gut|January 1, 1987
Do insulin and the insulin like growth factors (IGFs) stimulate growth of the exocrine pancreas?J Mössner, C D Logsdon, I D Goldfine, et al.
Molecular Pharmacology|September 1, 1995
Mutation of carboxyl-terminal threonine residues in human m3 muscarinic acetylcholine receptor modulates the extent of sequestration and desensitizationJ Yang, J A Williams, D I Yule, et al.
Diabetes|September 1, 1985
Insulin, via its own receptor, regulates growth and amylase synthesis in pancreatic acinar AR42J cellsJ Mössner, C D Logsdon, J A Williams, et al.
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