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C D Malis

Showing results (1-10 of 11) with videos related to

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Progress in Clinical and Biological Research|January 1, 1988
Susceptibility of mitochondrial membranes to calcium and reactive oxygen species: implications for ischemic and toxic tissue damageC D Malis, J V Bonventre
The Journal of Biological Chemistry|October 25, 1986
Mechanism of calcium potentiation of oxygen free radical injury to renal mitochondria. A model for post-ischemic and toxic mitochondrial damageC D Malis, J V Bonventre
Analytical Biochemistry|April 1, 1981
A rapid extraction technique for atomic absorption determinations of kidney calciumW P Tew, C D Malis, W G Walker
The Journal of Laboratory and Clinical Medicine|May 1, 1984
Nephrotoxicity of lysine and of a single dose of aminoglycoside in rats given lysineC D Malis, L C Racusen, K Solez, et al.
The American Journal of Physiology|December 1, 1983
Effects of verapamil in models of ischemic acute renal failure in the ratC D Malis, J Y Cheung, A Leaf, et al.
The New England Journal of Medicine|June 26, 1986
Calcium and ischemic injuryJ Y Cheung, J V Bonventre, C D Malis, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Incorporation of marine lipids into mitochondrial membranes increases susceptibility to damage by calcium and reactive oxygen species: evidence for enhanced activation of phospholipase A2 in mitochondria enriched with n-3 fatty acidsC D Malis, P C Weber, A Leaf, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 1, 1981
Phosphocitrate inhibits mitochondrial and cytosolic accumulation of calcium in kidney cells in vivoW P Tew, C D Malis, J E Howard, et al.
Circulation|September 1, 1991
Effects of dietary omega 3 fatty acids on vascular contractility in preanoxic and postanoxic aortic ringsC D Malis, A Leaf, G S Varadarajan, et al.
The American Journal of Physiology|October 1, 1989
n-3 fatty acids increase postischemic blood flow but do not reduce myocardial necrosisT Force, C D Malis, J L Guerrero, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Progress in Clinical and Biological Research|January 1, 1988
Susceptibility of mitochondrial membranes to calcium and reactive oxygen species: implications for ischemic and toxic tissue damageC D Malis, J V Bonventre
The Journal of Biological Chemistry|October 25, 1986
Mechanism of calcium potentiation of oxygen free radical injury to renal mitochondria. A model for post-ischemic and toxic mitochondrial damageC D Malis, J V Bonventre
Analytical Biochemistry|April 1, 1981
A rapid extraction technique for atomic absorption determinations of kidney calciumW P Tew, C D Malis, W G Walker
The Journal of Laboratory and Clinical Medicine|May 1, 1984
Nephrotoxicity of lysine and of a single dose of aminoglycoside in rats given lysineC D Malis, L C Racusen, K Solez, et al.
The American Journal of Physiology|December 1, 1983
Effects of verapamil in models of ischemic acute renal failure in the ratC D Malis, J Y Cheung, A Leaf, et al.
The New England Journal of Medicine|June 26, 1986
Calcium and ischemic injuryJ Y Cheung, J V Bonventre, C D Malis, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Incorporation of marine lipids into mitochondrial membranes increases susceptibility to damage by calcium and reactive oxygen species: evidence for enhanced activation of phospholipase A2 in mitochondria enriched with n-3 fatty acidsC D Malis, P C Weber, A Leaf, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 1, 1981
Phosphocitrate inhibits mitochondrial and cytosolic accumulation of calcium in kidney cells in vivoW P Tew, C D Malis, J E Howard, et al.
Circulation|September 1, 1991
Effects of dietary omega 3 fatty acids on vascular contractility in preanoxic and postanoxic aortic ringsC D Malis, A Leaf, G S Varadarajan, et al.
The American Journal of Physiology|October 1, 1989
n-3 fatty acids increase postischemic blood flow but do not reduce myocardial necrosisT Force, C D Malis, J L Guerrero, et al.
Pageof 2