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La Revue De Medecine Interne|April 21, 2007
[Hepcidin and iron metabolism]C Delaby, J-C Deybach, C BeaumontCellular and Molecular Biology (Noisy-Le-Grand, France)|March 10, 2009
Excessive erythrocyte PPIX influences the hematologic status and iron metabolism in patients with dominant erythropoietic protoporphyriaC Delaby, S Lyoumi, S Ducamp, et al.Journal of Internal Medicine|July 3, 2009
Role of two nutritional hepatic markers (insulin-like growth factor 1 and transthyretin) in the clinical assessment and follow-up of acute intermittent porphyria patientsC Delaby, J To-Figueras, J C Deybach, et al.American Journal of Human Genetics|May 1, 1984
Enzymatic and immunological studies of uroporphyrinogen decarboxylase in familial porphyria cutanea tarda and hepatoerythropoietic porphyriaH de Verneuil, C Beaumont, J C Deybach, et al.Experimental Cell Research|October 1, 1984
Effects of succinylacetone on dimethylsulfoxide-mediated induction of heme pathway enzymes in mouse friend virus-transformed erythroleukemia cellsC Beaumont, J C Deybach, B Grandchamp, et al.American Journal of Human Genetics|February 1, 1996
Modulation of the phenotype in dominant erythropoietic protoporphyria by a low expression of the normal ferrochelatase alleleL Gouya, J C Deybach, J Lamoril, et al.Nucleic Acids Research|August 25, 1989
A point mutation G----A in exon 12 of the porphobilinogen deaminase gene results in exon skipping and is responsible for acute intermittent porphyriaB Grandchamp, C Picat, F de Rooij, et al.Annales De Medecine Interne|January 1, 1993
[Acute attacks of hepatic porphyria: specific treatment with heme arginate]Y Nordmann, J C DeybachJournal of Bioenergetics and Biomembranes|April 1, 1995
Porphobilinogen deaminase gene structure and molecular defectsJ C Deybach, H PuyPageof 24