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C E Barry

Showing results (41-50 of 58) with videos related to

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The International Journal of Tuberculosis and Lung Disease : the Official Journal of the International Union Against Tuberculosis and Lung Disease|June 12, 2016
The impact of social conditions on patient adherence to pulmonary tuberculosis treatmentH Choi, H Chung, C Muntaner, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 3, 2000
The salicylate-derived mycobactin siderophores of Mycobacterium tuberculosis are essential for growth in macrophagesJ J De Voss, K Rutter, B G Schroeder, et al.
The Journal of Infectious Diseases|November 1, 1996
Biochemical and genetic data suggest that InhA is not the primary target for activated isoniazid in Mycobacterium tuberculosisK Mdluli, D R Sherman, M J Hickey, et al.
Progress in Lipid Research|November 26, 1998
Mycolic acids: structure, biosynthesis and physiological functionsC E Barry, R E Lee, K Mdluli, et al.
Science (New York, N.Y.)|June 14, 1996
Compensatory ahpC gene expression in isoniazid-resistant Mycobacterium tuberculosisD R Sherman, K Mdluli, M J Hickey, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 5, 1998
The 16-kDa alpha-crystallin (Acr) protein of Mycobacterium tuberculosis is required for growth in macrophagesY Yuan, D D Crane, R M Simpson, et al.
Journal of Natural Products|July 1, 1985
Desacetyluvaricin from Uvaria accuminata, configuration of uvaricin at C-36S D Jolad, J J Hoffmann, J R Cole, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 26, 2001
Virulence of a Mycobacterium tuberculosis clinical isolate in mice is determined by failure to induce Th1 type immunity and is associated with induction of IFN-alpha /betaC Manca, L Tsenova, A Bergtold, et al.
Science (New York, N.Y.)|June 11, 1998
Inhibition of a Mycobacterium tuberculosis beta-ketoacyl ACP synthase by isoniazidK Mdluli, R A Slayden, Y Zhu, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 3, 1999
Mycobacterium tuberculosis CDC1551 induces a more vigorous host response in vivo and in vitro, but is not more virulent than other clinical isolatesC Manca, L Tsenova, C E Barry, et al.
Pageof 6

Showing results (41-50 of 58) with videos related to

Sort By:
Pageof 6
The International Journal of Tuberculosis and Lung Disease : the Official Journal of the International Union Against Tuberculosis and Lung Disease|June 12, 2016
The impact of social conditions on patient adherence to pulmonary tuberculosis treatmentH Choi, H Chung, C Muntaner, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 3, 2000
The salicylate-derived mycobactin siderophores of Mycobacterium tuberculosis are essential for growth in macrophagesJ J De Voss, K Rutter, B G Schroeder, et al.
The Journal of Infectious Diseases|November 1, 1996
Biochemical and genetic data suggest that InhA is not the primary target for activated isoniazid in Mycobacterium tuberculosisK Mdluli, D R Sherman, M J Hickey, et al.
Progress in Lipid Research|November 26, 1998
Mycolic acids: structure, biosynthesis and physiological functionsC E Barry, R E Lee, K Mdluli, et al.
Science (New York, N.Y.)|June 14, 1996
Compensatory ahpC gene expression in isoniazid-resistant Mycobacterium tuberculosisD R Sherman, K Mdluli, M J Hickey, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 5, 1998
The 16-kDa alpha-crystallin (Acr) protein of Mycobacterium tuberculosis is required for growth in macrophagesY Yuan, D D Crane, R M Simpson, et al.
Journal of Natural Products|July 1, 1985
Desacetyluvaricin from Uvaria accuminata, configuration of uvaricin at C-36S D Jolad, J J Hoffmann, J R Cole, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 26, 2001
Virulence of a Mycobacterium tuberculosis clinical isolate in mice is determined by failure to induce Th1 type immunity and is associated with induction of IFN-alpha /betaC Manca, L Tsenova, A Bergtold, et al.
Science (New York, N.Y.)|June 11, 1998
Inhibition of a Mycobacterium tuberculosis beta-ketoacyl ACP synthase by isoniazidK Mdluli, R A Slayden, Y Zhu, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 3, 1999
Mycobacterium tuberculosis CDC1551 induces a more vigorous host response in vivo and in vitro, but is not more virulent than other clinical isolatesC Manca, L Tsenova, C E Barry, et al.
Pageof 6